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10.3947/ic.2017.49.1.10 http://scihub22266oqcxt.onion/10.3947/ic.2017.49.1.10 C5382045!5382045!28378540     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Infect+Chemother 2017 ; 49 (1): 10-21 Nephropedia Template TP {{tp|p=28378540|t=2017. Mitochondrial Dysfunction and Immune Cell Metabolism in Sepsis |pdf=|usr=}}{{28378540}} gab.com Text Mitochondrial Dysfunction and Immune Cell Metabolism in Sepsis JO: Infect Chemother http://www.kidney.de/pget.php?&tw=1&pmid=28378540 #FOAvip Sepsis is a life threatening condition mediated by systemic infection, but also triggered by hemorrhage and trauma. These are significant causes of organ injury implicated in morbidity and mortality, as well as post-sepsis complications associated with dysfunction of innate and adaptive immunity. The role of cellular bioenergetics and loss of metabolic plasticity of immune cells is increasingly emerging in the pathogenesis of sepsis. This review describes mitochondrial biology and metabolic alterations of immune cells due to sepsis, as well as indicates plausible therapeutic opportunities. Twit Text FOAVip Mitochondrial Dysfunction and Immune Cell Metabolism in Sepsis ????Infect Chemother http://www.kidney.de/pget.php?&tw=1&pmid=28378540 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28378540 #FOAvip English Wikipedia <ref>{{Cite pmid|28378540}}</ref> Mitochondrial Dysfunction and Immune Cell Metabolism in Sepsis #MMPMID28378540 Park DW; Zmijewski JW Infect Chemother 2017[Mar]; 49 (1): 10-21 PMID28378540show ga Sepsis is a life threatening condition mediated by systemic infection, but also triggered by hemorrhage and trauma. These are significant causes of organ injury implicated in morbidity and mortality, as well as post-sepsis complications associated with dysfunction of innate and adaptive immunity. The role of cellular bioenergetics and loss of metabolic plasticity of immune cells is increasingly emerging in the pathogenesis of sepsis. This review describes mitochondrial biology and metabolic alterations of immune cells due to sepsis, as well as indicates plausible therapeutic opportunities. äMitochondrial Dysfunction and Immune Cell Metabolism in Sepsis (epmc).pdf DeepDyve Pubget Overpricing