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10.1101/gr.213256.116 http://scihub22266oqcxt.onion/10.1101/gr.213256.116 C5378178!5378178!28073918     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28073918&cmd=llinks): Failed to open stream: HTTP request failed! 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Identical bacterial populations colonize premature infant gut, skin, and oral microbiomes and exhibit different in situ growth rates |pdf=|usr=}}{{28073918}} gab.com Text Identical bacterial populations colonize premature infant gut, skin, and oral microbiomes and exhibit different in situ growth rates JO: Genome Res http://www.kidney.de/pget.php?&tw=1&pmid=28073918 #FOAvip The initial microbiome impacts the health and future development of premature infants. Methodological limitations have led to gaps in our understanding of the habitat range and subpopulation complexity of founding strains, as well as how different body sites support microbial growth. Here, we used metagenomics to reconstruct genomes of strains that colonized the skin, mouth, and gut of two hospitalized premature infants during the first month of life. Seven bacterial populations, considered to be identical given whole-genome average nucleotide identity of >99.9%, colonized multiple body sites, yet none were shared between infants. Gut-associated Citrobacter koseri genomes harbored 47 polymorphic sites that we used to define 10 subpopulations, one of which appeared in the gut after 1 wk but did not spread to other body sites. Differential genome coverage was used to measure bacterial population replication rates in situ. In all cases where the same bacterial population was detected in multiple body sites, replication rates were faster in mouth and skin compared to the gut. The ability of identical strains to colonize multiple body sites underscores the habit flexibility of initial colonists, whereas differences in microbial replication rates between body sites suggest differences in host control and/or resource availability. Population genomic analyses revealed microdiversity within bacterial populations, implying initial inoculation by multiple individual cells with distinct genotypes. Overall, however, the overlap of strains across body sites implies that the premature infant microbiome can exhibit very low microbial diversity. Twit Text FOAVip Identical bacterial populations colonize premature infant gut, skin, and oral microbiomes and exhibit different in situ growth rates ????Genome Res http://www.kidney.de/pget.php?&tw=1&pmid=28073918 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28073918 #FOAvip English Wikipedia <ref>{{Cite pmid|28073918}}</ref> Identical bacterial populations colonize premature infant gut, skin, and oral microbiomes and exhibit different in situ growth rates #MMPMID28073918 Olm MR; Brown CT; Brooks B; Firek B; Baker R; Burstein D; Soenjoyo K; Thomas BC; Morowitz M; Banfield JF Genome Res 2017[Apr]; 27 (4): 601-12 PMID28073918show ga The initial microbiome impacts the health and future development of premature infants. Methodological limitations have led to gaps in our understanding of the habitat range and subpopulation complexity of founding strains, as well as how different body sites support microbial growth. Here, we used metagenomics to reconstruct genomes of strains that colonized the skin, mouth, and gut of two hospitalized premature infants during the first month of life. Seven bacterial populations, considered to be identical given whole-genome average nucleotide identity of >99.9%, colonized multiple body sites, yet none were shared between infants. Gut-associated Citrobacter koseri genomes harbored 47 polymorphic sites that we used to define 10 subpopulations, one of which appeared in the gut after 1 wk but did not spread to other body sites. Differential genome coverage was used to measure bacterial population replication rates in situ. In all cases where the same bacterial population was detected in multiple body sites, replication rates were faster in mouth and skin compared to the gut. The ability of identical strains to colonize multiple body sites underscores the habit flexibility of initial colonists, whereas differences in microbial replication rates between body sites suggest differences in host control and/or resource availability. Population genomic analyses revealed microdiversity within bacterial populations, implying initial inoculation by multiple individual cells with distinct genotypes. Overall, however, the overlap of strains across body sites implies that the premature infant microbiome can exhibit very low microbial diversity. äIdentical bacterial populations colonize premature infant gut, skin, a(epmc).pdf DeepDyve Pubget Overpricing