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10.21873/cgp.20026 http://scihub22266oqcxt.onion/10.21873/cgp.20026 C5369313!5369313 !28387653     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28387653 &cmd=llinks): Failed to open stream: HTTP request failed! 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Overexpression of microRNA-196b Accelerates Invasiveness of Cancer Cells in Recurrent Epithelial Ovarian Cancer Through Regulation of Homeobox A9 |pdf=|usr=}}{{28387653 }} gab.com Text Overexpression of microRNA-196b Accelerates Invasiveness of Cancer Cells in Recurrent Epithelial Ovarian Cancer Through Regulation of Homeobox A9 JO: Cancer Genomics Proteomics http://www.kidney.de/pget.php?&tw=1&pmid=28387653 #FOAvip BACKGROUND/AIM: Although microRNAs (miRNAs) are known to influence messenger RNA post-transcriptional control and contribute to human tumorigenesis, little is known about the differences in miRNA expression between primary and recurrent epithelial ovarian cancer (EOC). The purpose of this study was to assess the differential miRNA expression between primary and recurrent EOC and to investigate whether miR-196b could regulate the expression of the Homeobox A9 (HOXA9) gene, and thus affect the invasiveness of cancer cells in recurrent EOC. MATERIALS AND METHODS: Microarrays were used to generate the expression profiles of 6658 miRNAs from samples of 10 patients with EOC. miRNA expression patterns were compared between primary and recurrent EOC. Aberrantly expressed miRNA, associated genes, and invasion activities were validated by a luciferase assay and an in vitro invasion assay. RESULTS: miRNA microarray analysis identified 33 overexpressed miRNAs (including miR-196b) and 18 under expressed miRNAs in recurrent EOC from 6658 human miRNAs. HOXA9 expression was inversely correlated with miR-196b levels in recurrent EOC. We noted that miR-196b induced ovarian cancer cell invasiveness in recurrent EOC by an in vitro invasion assay. CONCLUSION: Overexpression of miR-196b may contribute to invasion activities in recurrent EOC by regulating the HOXA9 gene. Moreover, miR-196b can be a potential biomarker in recurrent EOC. Twit Text FOAVip Overexpression of microRNA-196b Accelerates Invasiveness of Cancer Cells in Recurrent Epithelial Ovarian Cancer Through Regulation of Homeobox A9 ????Cancer Genomics Proteomics http://www.kidney.de/pget.php?&tw=1&pmid=28387653 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28387653 #FOAvip English Wikipedia <ref>{{Cite pmid|28387653 }}</ref> Overexpression of microRNA-196b Accelerates Invasiveness of Cancer Cells in Recurrent Epithelial Ovarian Cancer Through Regulation of Homeobox A9 #MMPMID28387653 Chong GO ; Jeon HS ; Han HS ; Son JW ; Lee YH ; Hong DG ; Park HJ ; Lee YS ; Cho YL Cancer Genomics Proteomics 2017[Mar]; 14 (2 ): 137-141 PMID28387653 show ga BACKGROUND/AIM: Although microRNAs (miRNAs) are known to influence messenger RNA post-transcriptional control and contribute to human tumorigenesis, little is known about the differences in miRNA expression between primary and recurrent epithelial ovarian cancer (EOC). The purpose of this study was to assess the differential miRNA expression between primary and recurrent EOC and to investigate whether miR-196b could regulate the expression of the Homeobox A9 (HOXA9) gene, and thus affect the invasiveness of cancer cells in recurrent EOC. MATERIALS AND METHODS: Microarrays were used to generate the expression profiles of 6658 miRNAs from samples of 10 patients with EOC. miRNA expression patterns were compared between primary and recurrent EOC. Aberrantly expressed miRNA, associated genes, and invasion activities were validated by a luciferase assay and an in vitro invasion assay. RESULTS: miRNA microarray analysis identified 33 overexpressed miRNAs (including miR-196b) and 18 under expressed miRNAs in recurrent EOC from 6658 human miRNAs. HOXA9 expression was inversely correlated with miR-196b levels in recurrent EOC. We noted that miR-196b induced ovarian cancer cell invasiveness in recurrent EOC by an in vitro invasion assay. CONCLUSION: Overexpression of miR-196b may contribute to invasion activities in recurrent EOC by regulating the HOXA9 gene. Moreover, miR-196b can be a potential biomarker in recurrent EOC. |*Gene Expression Regulation, Neoplastic [MESH] |3' Untranslated Regions/genetics [MESH] |Biomarkers, Tumor/genetics [MESH] |Carcinoma, Ovarian Epithelial [MESH] |Cell Line, Tumor [MESH] |Cell Movement/genetics [MESH] |Female [MESH] |Gene Expression Profiling/*methods [MESH] |HEK293 Cells [MESH] |Homeodomain Proteins/*genetics [MESH] |Humans [MESH] |MicroRNAs/*genetics [MESH] |Neoplasm Invasiveness [MESH] |Neoplasm Recurrence, Local [MESH] |Neoplasms, Glandular and Epithelial/*genetics/pathology [MESH] |Oligonucleotide Array Sequence Analysis/methods [MESH] |Ovarian Neoplasms/*genetics/pathology [MESH] |Reverse Transcriptase Polymerase Chain Reaction [MESH]Overexpression of microRNA-196b Accelerates Invasiveness of Cancer Cel(epmc).pdf DeepDyve Pubget Overpricing