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10.1016/j.ijcha.2016.11.006 http://scihub22266oqcxt.onion/10.1016/j.ijcha.2016.11.006 C5368285!5368285!28382321     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Int+J+Cardiol+Heart+Vasc 2017 ; 14 (ä): 8-13 Nephropedia Template TP {{tp|p=28382321|t=2017. Electrophysiological mechanisms of long and short QT syndromes |pdf=|usr=}}{{28382321}} gab.com Text Electrophysiological mechanisms of long and short QT syndromes JO: Int J Cardiol Heart Vasc http://www.kidney.de/pget.php?&tw=1&pmid=28382321 #FOAvip The QT interval on the human electrocardiogram is normally in the order of 450 ms, and reflects the summated durations of action potential (AP) depolarization and repolarization of ventricular myocytes. Both prolongation and shortening in the QT interval have been associated with ventricular tachy-arrhythmias, which predispose affected individuals to sudden cardiac death. In this article, the molecular determinants of the AP duration and the causes of long and short QT syndromes (LQTS and SQTS) are explored. This is followed by a review of the recent advances on their arrhythmogenic mechanisms involving reentry and/or triggered activity based on experiments conducted in mouse models. Established and novel clinical risk markers based on the QT interval for the prediction of arrhythmic risk and cardiovascular mortality are presented here. It is concluded by a discussion on strategies for the future rational design of anti-arrhythmic agents. Twit Text FOAVip Electrophysiological mechanisms of long and short QT syndromes ????Int J Cardiol Heart Vasc http://www.kidney.de/pget.php?&tw=1&pmid=28382321 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28382321 #FOAvip English Wikipedia <ref>{{Cite pmid|28382321}}</ref> Electrophysiological mechanisms of long and short QT syndromes #MMPMID28382321 Tse G; Chan YWF; Keung W; Yan BP Int J Cardiol Heart Vasc 2017[Mar]; 14 (ä): 8-13 PMID28382321show ga The QT interval on the human electrocardiogram is normally in the order of 450 ms, and reflects the summated durations of action potential (AP) depolarization and repolarization of ventricular myocytes. Both prolongation and shortening in the QT interval have been associated with ventricular tachy-arrhythmias, which predispose affected individuals to sudden cardiac death. In this article, the molecular determinants of the AP duration and the causes of long and short QT syndromes (LQTS and SQTS) are explored. This is followed by a review of the recent advances on their arrhythmogenic mechanisms involving reentry and/or triggered activity based on experiments conducted in mouse models. Established and novel clinical risk markers based on the QT interval for the prediction of arrhythmic risk and cardiovascular mortality are presented here. It is concluded by a discussion on strategies for the future rational design of anti-arrhythmic agents. äElectrophysiological mechanisms of long and short QT syndromes (epmc).pdf DeepDyve Pubget Overpricing