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10.1371/journal.pone.0173906 http://scihub22266oqcxt.onion/10.1371/journal.pone.0173906 C5363828!5363828!28333979     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 267.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 267.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\28333979.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       PLoS+One 2017 ; 12 (3): ä Nephropedia Template TP {{tp|p=28333979|t=2017. The interaction between fluid status and angiopoietin-2 in adverse renal outcomes of chronic kidney disease |pdf=|usr=}}{{28333979}} gab.com Text The interaction between fluid status and angiopoietin-2 in adverse renal outcomes of chronic kidney disease JO: PLoS One http://www.kidney.de/pget.php?&tw=1&pmid=28333979 #FOAvip Background: Fluid overload is not only the characteristic but also an important complication in chronic kidney disease (CKD) patients. Angiopoietin-2 (Angpt2) disturbs endothelium and vessel permeability, which may induce fluid overload. The aim of this study is to examine the interaction between fluid status and Angpt2 in adverse renal outcomes of CKD. Methods: This cohort study enrolled 290 patients with CKD stages 3?5 from January 2011 to December 2011 and followed up until December 2015. Fluid status was presented as overhydration (OH) value measured by body composition monitor, while OH>1.1L was defined as fluid overload. Renal outcomes were defined as commencing dialysis and rapid renal function decline (the slope of estimated glomerular filtration rate < -5 ml/min/1.73 m2/y). Results: During a mean follow-up of 38.6±18.3 months, 125 (43.1%) patients progressed to commencing dialysis and 99(34.7%) patients presented rapid renal function decline. All patients were stratified by OH of 1.1L and the median of circulating Angpt2. These patients with both OH>1.1L and high circulating Angpt2 were more likely to reach commencing dialysis compared to other groups. The risks for commencing dialysis and rapid renal function decline were significantly higher in patients with OH>1.1L and high circulating Angpt2 level compared to those with OH?1.1L and low circulating Angpt2 (2.14, 1.21?3.78, P = 0.009; 4.96, 1.45?16.97, P = 0.01). There was a significant interaction between OH level and circulating Angpt2 in entering dialysis (P-interaction = 0.02). Conclusions: Fluid overload and Angpt2 might have a synergistic effect on adverse renal outcomes in CKD patients. Twit Text FOAVip The interaction between fluid status and angiopoietin-2 in adverse renal outcomes of chronic kidney disease ????PLoS One http://www.kidney.de/pget.php?&tw=1&pmid=28333979 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28333979 #FOAvip English Wikipedia <ref>{{Cite pmid|28333979}}</ref> The interaction between fluid status and angiopoietin-2 in adverse renal outcomes of chronic kidney disease #MMPMID28333979 Tsai YC; Chiu YW; Kuo HT; Lee JJ; Lee SC; Chen TH; Lin MY; Hwang SJ; Kuo MC; Hsu YL; Chen HC PLoS One 2017[]; 12 (3): ä PMID28333979show ga Background: Fluid overload is not only the characteristic but also an important complication in chronic kidney disease (CKD) patients. Angiopoietin-2 (Angpt2) disturbs endothelium and vessel permeability, which may induce fluid overload. The aim of this study is to examine the interaction between fluid status and Angpt2 in adverse renal outcomes of CKD. Methods: This cohort study enrolled 290 patients with CKD stages 3?5 from January 2011 to December 2011 and followed up until December 2015. Fluid status was presented as overhydration (OH) value measured by body composition monitor, while OH>1.1L was defined as fluid overload. Renal outcomes were defined as commencing dialysis and rapid renal function decline (the slope of estimated glomerular filtration rate < -5 ml/min/1.73 m2/y). Results: During a mean follow-up of 38.6±18.3 months, 125 (43.1%) patients progressed to commencing dialysis and 99(34.7%) patients presented rapid renal function decline. All patients were stratified by OH of 1.1L and the median of circulating Angpt2. These patients with both OH>1.1L and high circulating Angpt2 were more likely to reach commencing dialysis compared to other groups. The risks for commencing dialysis and rapid renal function decline were significantly higher in patients with OH>1.1L and high circulating Angpt2 level compared to those with OH?1.1L and low circulating Angpt2 (2.14, 1.21?3.78, P = 0.009; 4.96, 1.45?16.97, P = 0.01). There was a significant interaction between OH level and circulating Angpt2 in entering dialysis (P-interaction = 0.02). Conclusions: Fluid overload and Angpt2 might have a synergistic effect on adverse renal outcomes in CKD patients. äThe interaction between fluid status and angiopoietin-2 in adverse ren(epmc).pdf DeepDyve Pubget Overpricing