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10.1016/j.jpsychires.2016.12.016 http://scihub22266oqcxt.onion/10.1016/j.jpsychires.2016.12.016 C5362299!5362299 !28086129     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28086129 &cmd=llinks): Failed to open stream: HTTP request failed! 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Hyper-modulation of brain networks by the amygdala among women with Borderline Personality Disorder: Network signatures of affective interference during cognitive processing |pdf=|usr=}}{{28086129 }} gab.com Text Hyper-modulation of brain networks by the amygdala among women with Borderline Personality Disorder: Network signatures of affective interference during cognitive processing JO: J Psychiatr Res http://www.kidney.de/pget.php?&tw=1&pmid=28086129 #FOAvip Emotion dysregulation is a core characteristic of patients with Borderline Personality Disorder (BPD), and is often attributed to an imbalance in fronto-limbic network function. Hyperarousal of amygdala, especially in response to negative affective stimuli, results in affective interference with cognitive processing of executive functions. Clinical consequences include the impulsive-aggression, suicidal and self-injurious behaviors which characterize BPD. Dysfunctional interactions between amygdala and its network targets have not been well characterized during cognitive task performance. Using psychophysiological interaction analysis (PPI), we mapped network profiles of amygdala interaction with key regulatory regions during a Go No-Go task, modified to use negative, positive and neutral Ekman faces as targets. Fifty-six female subjects, 31 BPD and 25 healthy controls (HC), completed the affectively valenced Go No-Go task during fMRI scanning. In the negative affective condition, the amygdala exerted greater modulation of its targets in BPD compared to HC subjects in Rt. OFC, Rt. dACC, Rt. Parietal cortex, Rt. Basal Ganglia, and Rt. dlPFC. Across the spectrum of affective contrasts, hypermodulation in BPD subjects observed the following ordering: Negative > Neutral > Positive contrast. The amygdala seed exerted modulatory effects on specific target regions important in processing response inhibition and motor impulsiveness. The vulnerability of BPD subjects to affective interference with impulse control may be due to specific network dysfunction related to amygdala hyper-arousal and its effects on prefrontal regulatory regions such as the OFC and dACC. Twit Text FOAVip Hyper-modulation of brain networks by the amygdala among women with Borderline Personality Disorder: Network signatures of affective interference during cognitive processing ????J Psychiatr Res http://www.kidney.de/pget.php?&tw=1&pmid=28086129 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28086129 #FOAvip English Wikipedia <ref>{{Cite pmid|28086129 }}</ref> Hyper-modulation of brain networks by the amygdala among women with Borderline Personality Disorder: Network signatures of affective interference during cognitive processing #MMPMID28086129 Soloff PH ; Abraham K ; Ramaseshan K ; Burgess A ; Diwadkar VA J Psychiatr Res 2017[May]; 88 (ä): 56-63 PMID28086129 show ga Emotion dysregulation is a core characteristic of patients with Borderline Personality Disorder (BPD), and is often attributed to an imbalance in fronto-limbic network function. Hyperarousal of amygdala, especially in response to negative affective stimuli, results in affective interference with cognitive processing of executive functions. Clinical consequences include the impulsive-aggression, suicidal and self-injurious behaviors which characterize BPD. Dysfunctional interactions between amygdala and its network targets have not been well characterized during cognitive task performance. Using psychophysiological interaction analysis (PPI), we mapped network profiles of amygdala interaction with key regulatory regions during a Go No-Go task, modified to use negative, positive and neutral Ekman faces as targets. Fifty-six female subjects, 31 BPD and 25 healthy controls (HC), completed the affectively valenced Go No-Go task during fMRI scanning. In the negative affective condition, the amygdala exerted greater modulation of its targets in BPD compared to HC subjects in Rt. OFC, Rt. dACC, Rt. Parietal cortex, Rt. Basal Ganglia, and Rt. dlPFC. Across the spectrum of affective contrasts, hypermodulation in BPD subjects observed the following ordering: Negative > Neutral > Positive contrast. The amygdala seed exerted modulatory effects on specific target regions important in processing response inhibition and motor impulsiveness. The vulnerability of BPD subjects to affective interference with impulse control may be due to specific network dysfunction related to amygdala hyper-arousal and its effects on prefrontal regulatory regions such as the OFC and dACC. |*Brain Mapping [MESH] |Adolescent [MESH] |Adult [MESH] |Amygdala/diagnostic imaging/*physiopathology [MESH] |Borderline Personality Disorder/diagnostic imaging/*pathology [MESH] |Cognition/*physiology [MESH] |Female [MESH] |Humans [MESH] |Image Processing, Computer-Assisted [MESH] |Inhibition, Psychological [MESH] |Magnetic Resonance Imaging [MESH] |Middle Aged [MESH] |Mood Disorders/*etiology/pathology [MESH] |Neural Pathways/diagnostic imaging/*physiopathology [MESH] |Oxygen/blood [MESH] |Psychophysics [MESH] |Reflex, Startle/physiology [MESH]Hyper-modulation of brain networks by the amygdala among women with Bo(epmc).pdf DeepDyve Pubget Overpricing