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10.3389/fimmu.2017.00311 http://scihub22266oqcxt.onion/10.3389/fimmu.2017.00311 C5359241!5359241 !28377767     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28377767 &cmd=llinks): Failed to open stream: HTTP request failed! 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Immunometabolic Regulation of Interleukin-17-Producing T Helper Cells: Uncoupling New Targets for Autoimmunity |pdf=|usr=}}{{28377767 }} gab.com Text Immunometabolic Regulation of Interleukin-17-Producing T Helper Cells: Uncoupling New Targets for Autoimmunity JO: Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=28377767 #FOAvip Interleukin-17-producing T helper (Th17) cells are critical for the host defense of bacterial and fungal pathogens and also play a major role in driving pathogenic autoimmune responses. Recent studies have indicated that the generation of Th17?cells from naïve CD4(+) T cells is coupled with massive cellular metabolic adaptations, necessary to cope with different energy and metabolite requirements associated with switching from a resting to proliferative state. Furthermore, Th17?cells have to secure these metabolic adaptations when facing nutrient-limiting environments, such as at the sites of inflammation. Accumulating data indicates that this metabolic reprogramming is significantly linked to the differentiation of T helper cells and, particularly, that the metabolic changes of Th17?cells and anti-inflammatory Forkhead box P3(+) regulatory T cells are tightly and reciprocally regulated. Thus, a better understanding of these processes could offer potential new targets for therapeutic interventions for autoimmune diseases. In this mini-review, we will highlight some of the recent advances and discoveries in the field, with a particular focus on metabolic demands of Th17?cells and their implications for autoimmunity. Twit Text FOAVip Immunometabolic Regulation of Interleukin-17-Producing T Helper Cells: Uncoupling New Targets for Autoimmunity ????Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=28377767 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28377767 #FOAvip English Wikipedia <ref>{{Cite pmid|28377767 }}</ref> Immunometabolic Regulation of Interleukin-17-Producing T Helper Cells: Uncoupling New Targets for Autoimmunity #MMPMID28377767 Binger KJ ; Côrte-Real BF ; Kleinewietfeld M Front Immunol 2017[]; 8 (ä): 311 PMID28377767 show ga Interleukin-17-producing T helper (Th17) cells are critical for the host defense of bacterial and fungal pathogens and also play a major role in driving pathogenic autoimmune responses. Recent studies have indicated that the generation of Th17?cells from naïve CD4(+) T cells is coupled with massive cellular metabolic adaptations, necessary to cope with different energy and metabolite requirements associated with switching from a resting to proliferative state. Furthermore, Th17?cells have to secure these metabolic adaptations when facing nutrient-limiting environments, such as at the sites of inflammation. Accumulating data indicates that this metabolic reprogramming is significantly linked to the differentiation of T helper cells and, particularly, that the metabolic changes of Th17?cells and anti-inflammatory Forkhead box P3(+) regulatory T cells are tightly and reciprocally regulated. Thus, a better understanding of these processes could offer potential new targets for therapeutic interventions for autoimmune diseases. In this mini-review, we will highlight some of the recent advances and discoveries in the field, with a particular focus on metabolic demands of Th17?cells and their implications for autoimmunity. äImmunometabolic Regulation of Interleukin-17-Producing T Helper Cells:(epmc).pdf DeepDyve Pubget Overpricing