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10.1002/ece3.2818 http://scihub22266oqcxt.onion/10.1002/ece3.2818 C5355186!5355186!28331600     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Ecol+Evol 2017 ; 7 (6): 1936-41 Nephropedia Template TP {{tp|p=28331600|t=2017. Bacterial cell?to?cell signaling promotes the evolution of resistance to parasitic bacteriophages |pdf=|usr=}}{{28331600}} gab.com Text Bacterial cell to cell signaling promotes the evolution of resistance to parasitic bacteriophages JO: Ecol Evol http://www.kidney.de/pget.php?&tw=1&pmid=28331600 #FOAvip The evolution of host?parasite interactions could be affected by intraspecies variation between different host and parasite genotypes. Here we studied how bacterial host cell?to?cell signaling affects the interaction with parasites using two bacteria?specific viruses (bacteriophages) and the host bacterium Pseudomonas aeruginosa that communicates by secreting and responding to quorum sensing (QS) signal molecules. We found that a QS?signaling proficient strain was able to evolve higher levels of resistance to phages during a short?term selection experiment. This was unlikely driven by demographic effects (mutation supply and encounter rates), as nonsignaling strains reached higher population densities in the absence of phages in our selective environment. Instead, the evolved nonsignaling strains suffered relatively higher growth reduction in the absence of the phage, which could have constrained the phage resistance evolution. Complementation experiments with synthetic signal molecules showed that the Pseudomonas quinolone signal (PQS) improved the growth of nonsignaling bacteria in the presence of a phage, while the activation of las and rhl quorum sensing systems had no effect. Together, these results suggest that QS?signaling can promote the evolution of phage resistance and that the loss of QS?signaling could be costly in the presence of phages. Phage?bacteria interactions could therefore indirectly shape the evolution of intraspecies social interactions and PQS?mediated virulence in P. aeruginosa. Twit Text FOAVip Bacterial cell to cell signaling promotes the evolution of resistance to parasitic bacteriophages ????Ecol Evol http://www.kidney.de/pget.php?&tw=1&pmid=28331600 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28331600 #FOAvip English Wikipedia <ref>{{Cite pmid|28331600}}</ref> Bacterial cell?to?cell signaling promotes the evolution of resistance to parasitic bacteriophages #MMPMID28331600 Moreau P; Diggle SP; Friman V Ecol Evol 2017[Mar]; 7 (6): 1936-41 PMID28331600show ga The evolution of host?parasite interactions could be affected by intraspecies variation between different host and parasite genotypes. Here we studied how bacterial host cell?to?cell signaling affects the interaction with parasites using two bacteria?specific viruses (bacteriophages) and the host bacterium Pseudomonas aeruginosa that communicates by secreting and responding to quorum sensing (QS) signal molecules. We found that a QS?signaling proficient strain was able to evolve higher levels of resistance to phages during a short?term selection experiment. This was unlikely driven by demographic effects (mutation supply and encounter rates), as nonsignaling strains reached higher population densities in the absence of phages in our selective environment. Instead, the evolved nonsignaling strains suffered relatively higher growth reduction in the absence of the phage, which could have constrained the phage resistance evolution. Complementation experiments with synthetic signal molecules showed that the Pseudomonas quinolone signal (PQS) improved the growth of nonsignaling bacteria in the presence of a phage, while the activation of las and rhl quorum sensing systems had no effect. Together, these results suggest that QS?signaling can promote the evolution of phage resistance and that the loss of QS?signaling could be costly in the presence of phages. Phage?bacteria interactions could therefore indirectly shape the evolution of intraspecies social interactions and PQS?mediated virulence in P. aeruginosa. äBacterial cell?to?cell signaling promotes the evolution of resistance (epmc).pdf DeepDyve Pubget Overpricing