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10.18632/oncotarget.13783 http://scihub22266oqcxt.onion/10.18632/oncotarget.13783 C5352190!5352190!27926535     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Oncotarget 2017 ; 8 (1): 70-82 Nephropedia Template TP {{tp|p=27926535|t=2017. High salt diet stimulates gut Th17 response and exacerbates TNBS-induced colitis in mice |pdf=|usr=}}{{27926535}} gab.com Text High salt diet stimulates gut Th17 response and exacerbates TNBS-induced colitis in mice JO: Oncotarget http://www.kidney.de/pget.php?&tw=1&pmid=27926535 #FOAvip This study focuses on characterizing the effect of a high salt diet (HSD) on intestinal immunity and the risk of inflammatory bowel diseases (IBD). We found that mice on a HSD had an increased frequency of IL-17A producing cells in the intestinal lamina propria (LP) compared to mice on a normal diet (ND). Furthermore, most intestinal IL-17A producing cells were CD4+TCR?+ cells. A HSD increased the LP T helper 17 (Th17) responses in both the small and large intestines but did not increase the Th17 response of other gut-associated lymphoid organ. Although, HSD did not change the percentage of regulatory T (Treg) cells, HSD significantly inhibit secretion of IL-10 and the suppressive function of Treg cells. Moreover, we found that HSD exacerbates trinitrobenzenesulfonic acid (TNBS) induced colitis, and Th17 response was significantly increased in the colonic LP of HSD TNBS-treated mice compared with the ND TNBS-treated mice. This study demonstrates that HSD stimulates the intestinal Th17 response but inhibits the function of Treg cells. Moreover, HSD exacerbates TNBS induced mice colitis, suggesting that HSD disrupts the intestinal immunity and increases the risk of IBD. Twit Text FOAVip High salt diet stimulates gut Th17 response and exacerbates TNBS-induced colitis in mice ????Oncotarget http://www.kidney.de/pget.php?&tw=1&pmid=27926535 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27926535 #FOAvip English Wikipedia <ref>{{Cite pmid|27926535}}</ref> High salt diet stimulates gut Th17 response and exacerbates TNBS-induced colitis in mice #MMPMID27926535 Wei Y; Lu C; Chen J; Cui G; Wang L; Yu T; Yang Y; Wu W; Ding Y; Li L; Uede T; Chen Z; Diao H Oncotarget 2017[Jan]; 8 (1): 70-82 PMID27926535show ga This study focuses on characterizing the effect of a high salt diet (HSD) on intestinal immunity and the risk of inflammatory bowel diseases (IBD). We found that mice on a HSD had an increased frequency of IL-17A producing cells in the intestinal lamina propria (LP) compared to mice on a normal diet (ND). Furthermore, most intestinal IL-17A producing cells were CD4+TCR?+ cells. A HSD increased the LP T helper 17 (Th17) responses in both the small and large intestines but did not increase the Th17 response of other gut-associated lymphoid organ. Although, HSD did not change the percentage of regulatory T (Treg) cells, HSD significantly inhibit secretion of IL-10 and the suppressive function of Treg cells. Moreover, we found that HSD exacerbates trinitrobenzenesulfonic acid (TNBS) induced colitis, and Th17 response was significantly increased in the colonic LP of HSD TNBS-treated mice compared with the ND TNBS-treated mice. This study demonstrates that HSD stimulates the intestinal Th17 response but inhibits the function of Treg cells. Moreover, HSD exacerbates TNBS induced mice colitis, suggesting that HSD disrupts the intestinal immunity and increases the risk of IBD. äHigh salt diet stimulates gut Th17 response and exacerbates TNBS-induc(epmc).pdf DeepDyve Pubget Overpricing