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10.3389/fimmu.2017.00258 http://scihub22266oqcxt.onion/10.3389/fimmu.2017.00258 C5348535!5348535!28352268     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Front+Immunol 2017 ; 8 (ä): ä Nephropedia Template TP {{tp|p=28352268|t=2017. Type I and III Interferon in the Gut: Tight Balance between Host Protection and Immunopathology |pdf=|usr=}}{{28352268}} gab.com Text Type I and III Interferon in the Gut: Tight Balance between Host Protection and Immunopathology JO: Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=28352268 #FOAvip The intestinal mucosa forms an active interface to the outside word, facilitating nutrient and water uptake and at the same time acts as a barrier toward the highly colonized intestinal lumen. A tight balance of the mucosal immune system is essential to tolerate harmless antigens derived from food or commensals and to effectively defend against potentially dangerous pathogens. Interferons (IFN) provide a first line of host defense when cells detect an invading organism. Whereas type I IFN were discovered almost 60?years ago, type III IFN were only identified in the early 2000s. It was initially thought that type I IFN and type III IFN performed largely redundant functions. However, it is becoming increasingly clear that type III IFN exert distinct and non-redundant functions compared to type I IFN, especially in mucosal tissues. Here, we review recent progress made in unraveling the role of type I/III IFN in intestinal mucosal tissue in the steady state, in response to mucosal pathogens and during inflammation. Twit Text FOAVip Type I and III Interferon in the Gut: Tight Balance between Host Protection and Immunopathology ????Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=28352268 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28352268 #FOAvip English Wikipedia <ref>{{Cite pmid|28352268}}</ref> Type I and III Interferon in the Gut: Tight Balance between Host Protection and Immunopathology #MMPMID28352268 Pott J; Stockinger S Front Immunol 2017[]; 8 (ä): ä PMID28352268show ga The intestinal mucosa forms an active interface to the outside word, facilitating nutrient and water uptake and at the same time acts as a barrier toward the highly colonized intestinal lumen. A tight balance of the mucosal immune system is essential to tolerate harmless antigens derived from food or commensals and to effectively defend against potentially dangerous pathogens. Interferons (IFN) provide a first line of host defense when cells detect an invading organism. Whereas type I IFN were discovered almost 60?years ago, type III IFN were only identified in the early 2000s. It was initially thought that type I IFN and type III IFN performed largely redundant functions. However, it is becoming increasingly clear that type III IFN exert distinct and non-redundant functions compared to type I IFN, especially in mucosal tissues. Here, we review recent progress made in unraveling the role of type I/III IFN in intestinal mucosal tissue in the steady state, in response to mucosal pathogens and during inflammation. äType I and III Interferon in the Gut: Tight Balance between Host Prote(epmc).pdf DeepDyve Pubget Overpricing