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10.3389/fcimb.2017.00055 http://scihub22266oqcxt.onion/10.3389/fcimb.2017.00055 C5346588!5346588 !28348979     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28348979 &cmd=llinks): Failed to open stream: HTTP request failed! 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Biology of Acinetobacter baumannii: Pathogenesis, Antibiotic Resistance Mechanisms, and Prospective Treatment Options |pdf=|usr=}}{{28348979 }} gab.com Text Biology of Acinetobacter baumannii: Pathogenesis, Antibiotic Resistance Mechanisms, and Prospective Treatment Options JO: Front Cell Infect Microbiol http://www.kidney.de/pget.php?&tw=1&pmid=28348979 #FOAvip Acinetobacter baumannii is undoubtedly one of the most successful pathogens responsible for hospital-acquired nosocomial infections in the modern healthcare system. Due to the prevalence of infections and outbreaks caused by multi-drug resistant A. baumannii, few antibiotics are effective for treating infections caused by this pathogen. To overcome this problem, knowledge of the pathogenesis and antibiotic resistance mechanisms of A. baumannii is important. In this review, we summarize current studies on the virulence factors that contribute to A. baumannii pathogenesis, including porins, capsular polysaccharides, lipopolysaccharides, phospholipases, outer membrane vesicles, metal acquisition systems, and protein secretion systems. Mechanisms of antibiotic resistance of this organism, including acquirement of ?-lactamases, up-regulation of multidrug efflux pumps, modification of aminoglycosides, permeability defects, and alteration of target sites, are also discussed. Lastly, novel prospective treatment options for infections caused by multi-drug resistant A. baumannii are summarized. Twit Text FOAVip Biology of Acinetobacter baumannii: Pathogenesis, Antibiotic Resistance Mechanisms, and Prospective Treatment Options ????Front Cell Infect Microbiol http://www.kidney.de/pget.php?&tw=1&pmid=28348979 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28348979 #FOAvip English Wikipedia <ref>{{Cite pmid|28348979 }}</ref> Biology of Acinetobacter baumannii: Pathogenesis, Antibiotic Resistance Mechanisms, and Prospective Treatment Options #MMPMID28348979 Lee CR ; Lee JH ; Park M ; Park KS ; Bae IK ; Kim YB ; Cha CJ ; Jeong BC ; Lee SH Front Cell Infect Microbiol 2017[]; 7 (ä): 55 PMID28348979 show ga Acinetobacter baumannii is undoubtedly one of the most successful pathogens responsible for hospital-acquired nosocomial infections in the modern healthcare system. Due to the prevalence of infections and outbreaks caused by multi-drug resistant A. baumannii, few antibiotics are effective for treating infections caused by this pathogen. To overcome this problem, knowledge of the pathogenesis and antibiotic resistance mechanisms of A. baumannii is important. In this review, we summarize current studies on the virulence factors that contribute to A. baumannii pathogenesis, including porins, capsular polysaccharides, lipopolysaccharides, phospholipases, outer membrane vesicles, metal acquisition systems, and protein secretion systems. Mechanisms of antibiotic resistance of this organism, including acquirement of ?-lactamases, up-regulation of multidrug efflux pumps, modification of aminoglycosides, permeability defects, and alteration of target sites, are also discussed. Lastly, novel prospective treatment options for infections caused by multi-drug resistant A. baumannii are summarized. |*Drug Resistance, Multiple, Bacterial [MESH] |Acinetobacter Infections/*drug therapy [MESH] |Acinetobacter baumannii/*drug effects/*pathogenicity [MESH] |Animals [MESH] |Biological Therapy/methods [MESH] |Humans [MESH]Biology of Acinetobacter baumannii: Pathogenesis, Antibiotic Resistanc(epmc).pdf DeepDyve Pubget Overpricing