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10.1155/2017/7151285 http://scihub22266oqcxt.onion/10.1155/2017/7151285 C5340942!5340942 !28321152     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28321152 &cmd=llinks): Failed to open stream: HTTP request failed! 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Th1/Th17-Related Cytokines and Chemokines and Their Implications in the Pathogenesis of Pemphigus Vulgaris |pdf=|usr=}}{{28321152 }} gab.com Text Th1/Th17-Related Cytokines and Chemokines and Their Implications in the Pathogenesis of Pemphigus Vulgaris JO: Mediators Inflamm http://www.kidney.de/pget.php?&tw=1&pmid=28321152 #FOAvip Pemphigus vulgaris (PV) is an autoimmune disease characterized by the presence of IgG autoantibodies against desmoglein-3. Despite the variety of findings, the chemokine and cytokine profiles that characterize the immune response in the disease are still poorly explored. Thus, 20 PV patients and 20 controls were grouped according to gender, ethnicity, place of residence, and clinical parameters of the disease. Then, the levels of chemokines and of Th1/Th2/Th17/Treg/Th9/Th22-related cytokines were assessed in the serum. PV patients had higher levels of inflammatory Th1/Th17 cytokines (IFN-?, IL-17, and IL-23), as well as higher levels of CXCL8 and reduced levels of Th1/Th2-related chemokines (IP-10 and CCL11). However, no differences in the levels of IL-2, IL-6, TNF-?, IL-1?, IL-4, IL-9, IL-12, TGF-?, IL-33, MCP-1, RANTES, and MIP-1? were found between PV patients and their control counterparts. Furthermore, PV patients with skin lesions had higher serum levels of IL-6 and CXCL8 when compared to PV patients without lesions. Taken together, our findings describe the role of cytokines and chemokines associated with Th1/Th17 immune response in PV patients. Finally, these data are important for better understanding of the immune aspects that control disease outcome, and they may also provide important information about why patients develop autoantibodies against desmogleins. Twit Text FOAVip Th1/Th17-Related Cytokines and Chemokines and Their Implications in the Pathogenesis of Pemphigus Vulgaris ????Mediators Inflamm http://www.kidney.de/pget.php?&tw=1&pmid=28321152 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28321152 #FOAvip English Wikipedia <ref>{{Cite pmid|28321152 }}</ref> Th1/Th17-Related Cytokines and Chemokines and Their Implications in the Pathogenesis of Pemphigus Vulgaris #MMPMID28321152 Timoteo RP ; da Silva MV ; Miguel CB ; Silva DA ; Catarino JD ; Rodrigues Junior V ; Sales-Campos H ; Freire Oliveira CJ Mediators Inflamm 2017[]; 2017 (ä): 7151285 PMID28321152 show ga Pemphigus vulgaris (PV) is an autoimmune disease characterized by the presence of IgG autoantibodies against desmoglein-3. Despite the variety of findings, the chemokine and cytokine profiles that characterize the immune response in the disease are still poorly explored. Thus, 20 PV patients and 20 controls were grouped according to gender, ethnicity, place of residence, and clinical parameters of the disease. Then, the levels of chemokines and of Th1/Th2/Th17/Treg/Th9/Th22-related cytokines were assessed in the serum. PV patients had higher levels of inflammatory Th1/Th17 cytokines (IFN-?, IL-17, and IL-23), as well as higher levels of CXCL8 and reduced levels of Th1/Th2-related chemokines (IP-10 and CCL11). However, no differences in the levels of IL-2, IL-6, TNF-?, IL-1?, IL-4, IL-9, IL-12, TGF-?, IL-33, MCP-1, RANTES, and MIP-1? were found between PV patients and their control counterparts. Furthermore, PV patients with skin lesions had higher serum levels of IL-6 and CXCL8 when compared to PV patients without lesions. Taken together, our findings describe the role of cytokines and chemokines associated with Th1/Th17 immune response in PV patients. Finally, these data are important for better understanding of the immune aspects that control disease outcome, and they may also provide important information about why patients develop autoantibodies against desmogleins. |Adult [MESH] |Chemokine CCL3/metabolism [MESH] |Chemokine CCL5/metabolism [MESH] |Chemokines/*metabolism [MESH] |Cytokines/*metabolism [MESH] |Female [MESH] |Humans [MESH] |Interleukin-2/metabolism [MESH] |Interleukin-23/metabolism [MESH] |Interleukin-6/metabolism [MESH] |Interleukin-8/metabolism [MESH] |Male [MESH] |Middle Aged [MESH] |Pemphigus/*pathology [MESH] |T-Lymphocytes, Regulatory/metabolism [MESH] |Th1 Cells/*metabolism [MESH] |Th17 Cells/*metabolism [MESH] |Th2 Cells/metabolism [MESH] |Tumor Necrosis Factor-alpha/metabolism [MESH]Th1/Th17-Related Cytokines and Chemokines and Their Implications in th(epmc).pdf DeepDyve Pubget Overpricing