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10.1038/nri3532 http://scihub22266oqcxt.onion/10.1038/nri3532 C5340150!5340150!24064518     free     free     free       Nat+Rev+Immunol 2013 ; 13 (10): 722-37 Nephropedia Template TP {{tp|p=24064518|t=2013. Autophagy in infection, inflammation, and immunity |pdf=|usr=}}{{24064518}} gab.com Text Autophagy in infection, inflammation, and immunity JO: Nat Rev Immunol http://www.kidney.de/pget.php?&tw=1&pmid=24064518 #FOAvip Autophagy is a fundamental cell biological pathway affecting immunity. Whereas autophagy is an antimicrobial effector of conventional pattern recognition receptors (PRRs), autophagic adaptors termed SLRs represent a new subset of PRRs and provide the mechanistic basis for autophagic elimination of intracellular microbes. Autophagy controls inflammation via regulatory interactions with innate immunity signalling, by removing endogenous inflammasome agonists, and thorough effects on secretion of immune mediators. Autophagy contributes to antigen presentation, T cell homeostasis, and affects T cell repertories and polarization including Th17 inflammation. Here, we review the above relationships organized into four principal roles of autophagy in infection, inflammation, and immunity. Twit Text FOAVip Autophagy in infection, inflammation, and immunity ????Nat Rev Immunol http://www.kidney.de/pget.php?&tw=1&pmid=24064518 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24064518 #FOAvip English Wikipedia <ref>{{Cite pmid|24064518}}</ref> Autophagy in infection, inflammation, and immunity #MMPMID24064518 Deretic V; Saitoh T; Akira S Nat Rev Immunol 2013[Oct]; 13 (10): 722-37 PMID24064518show ga Autophagy is a fundamental cell biological pathway affecting immunity. Whereas autophagy is an antimicrobial effector of conventional pattern recognition receptors (PRRs), autophagic adaptors termed SLRs represent a new subset of PRRs and provide the mechanistic basis for autophagic elimination of intracellular microbes. Autophagy controls inflammation via regulatory interactions with innate immunity signalling, by removing endogenous inflammasome agonists, and thorough effects on secretion of immune mediators. Autophagy contributes to antigen presentation, T cell homeostasis, and affects T cell repertories and polarization including Th17 inflammation. Here, we review the above relationships organized into four principal roles of autophagy in infection, inflammation, and immunity. äAutophagy in infection, inflammation, and immunity (epmc).pdf DeepDyve Pubget Overpricing