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10.14740/jocmr2761w http://scihub22266oqcxt.onion/10.14740/jocmr2761w C5330765!5330765!28270882     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28270882&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Clin+Med+Res 2017 ; 9 (4): 233-47 Nephropedia Template TP {{tp|p=28270882|t=2017. Insulin-Like Growth Factor-1 Deficiency and Cirrhosis Establishment |pdf=|usr=}}{{28270882}} gab.com Text Insulin-Like Growth Factor-1 Deficiency and Cirrhosis Establishment JO: J Clin Med Res http://www.kidney.de/pget.php?&tw=1&pmid=28270882 #FOAvip Cirrhosis represents the final stage of chronic liver damage, which can be due to different factors such as alcohol, metabolic syndrome with liver steatosis, autoimmune diseases, drugs, toxins, and viral infection, among others. Nowadays, cirrhosis is an important health problem and it is an increasing cause of morbidity and mortality, being the 14th most common cause of death worldwide. The physiopathological pathways that lead to fibrosis and finally cirrhosis partly depend on the etiology. Nevertheless, some common features are shared in this complex mechanism. Recently, it has been demonstrated that cirrhosis is a dynamic process that can be altered in order to delay or revert fibrosis. In addition, when cirrhosis has been established, insulin-like growth factor-1 (IGF-1) deficiency or reduced availability is a common condition, independently of the etiology of chronic liver damage that leads to cirrhosis. IGF-1 deprivation seriously contributes to the progressive malnutrition of cirrhotic patient, increasing the vulnerability of the liver to establish an inflammatory and oxidative microenvironment with mitochondrial dysfunction. In this context, IGF-1 deficiency in cirrhotic patients can justify some of the common characteristics of these individuals. Several studies in animals and humans have been done in order to test the replacement of IGF-1 as a possible therapeutic option, with promising results. Twit Text FOAVip Insulin-Like Growth Factor-1 Deficiency and Cirrhosis Establishment ????J Clin Med Res http://www.kidney.de/pget.php?&tw=1&pmid=28270882 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28270882 #FOAvip English Wikipedia <ref>{{Cite pmid|28270882}}</ref> Insulin-Like Growth Factor-1 Deficiency and Cirrhosis Establishment #MMPMID28270882 de la Garza RG; Morales-Garza LA; Martin-Estal I; Castilla-Cortazar I J Clin Med Res 2017[Apr]; 9 (4): 233-47 PMID28270882show ga Cirrhosis represents the final stage of chronic liver damage, which can be due to different factors such as alcohol, metabolic syndrome with liver steatosis, autoimmune diseases, drugs, toxins, and viral infection, among others. Nowadays, cirrhosis is an important health problem and it is an increasing cause of morbidity and mortality, being the 14th most common cause of death worldwide. The physiopathological pathways that lead to fibrosis and finally cirrhosis partly depend on the etiology. Nevertheless, some common features are shared in this complex mechanism. Recently, it has been demonstrated that cirrhosis is a dynamic process that can be altered in order to delay or revert fibrosis. In addition, when cirrhosis has been established, insulin-like growth factor-1 (IGF-1) deficiency or reduced availability is a common condition, independently of the etiology of chronic liver damage that leads to cirrhosis. IGF-1 deprivation seriously contributes to the progressive malnutrition of cirrhotic patient, increasing the vulnerability of the liver to establish an inflammatory and oxidative microenvironment with mitochondrial dysfunction. In this context, IGF-1 deficiency in cirrhotic patients can justify some of the common characteristics of these individuals. Several studies in animals and humans have been done in order to test the replacement of IGF-1 as a possible therapeutic option, with promising results. äInsulin-Like Growth Factor-1 Deficiency and Cirrhosis Establishment (epmc).pdf DeepDyve Pubget Overpricing