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10.1111/jcpe.12664 http://scihub22266oqcxt.onion/10.1111/jcpe.12664 C5328907!5328907 !27978598     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27978598 &cmd=llinks): Failed to open stream: HTTP request failed! 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The subgingival microbiome, systemic inflammation and insulin resistance: The Oral Infections, Glucose Intolerance and Insulin Resistance Study |pdf=|usr=}}{{27978598 }} gab.com Text The subgingival microbiome, systemic inflammation and insulin resistance: The Oral Infections, Glucose Intolerance and Insulin Resistance Study JO: J Clin Periodontol http://www.kidney.de/pget.php?&tw=1&pmid=27978598 #FOAvip BACKGROUND: Inflammation might link microbial exposures to insulin resistance. We investigated the cross-sectional association between periodontal microbiota, inflammation and insulin resistance. METHODS: The Oral Infections, Glucose Intolerance and Insulin Resistance Study (ORIGINS) enrolled 152 diabetes-free adults (77% female) aged 20-55 years (mean = 34 ± 10). Three hundred and four subgingival plaque samples were analysed using the Human Oral Microbe Identification Microarray to measure the relative abundances of 379 taxa. C-reactive protein, interleukin-6, tumour necrosis factor-? and adiponectin were assessed from venous blood and their z-scores were summed to create an inflammatory score (IS). Insulin resistance was defined via the HOMA-IR. Associations between the microbiota and both inflammation and HOMA-IR were explored using multivariable linear regressions; mediation analyses assessed the proportion of the association explained by inflammation. RESULTS: The IS was inversely associated with Actinobacteria and Proteobacteria and positively associated with Firmicutes and TM7 (p-values < 0.05). Proteobacteria levels were associated with insulin resistance (p < 0.05). Inflammation explained 30-98% of the observed associations between levels of Actinobacteria, Proteobacteria or Firmicutes and insulin resistance (p-values < 0.05). Eighteen individual taxa were associated with inflammation (p < 0.05) and 22 with insulin resistance (p < 0.05). No findings for individual taxa met Bonferroni-adjusted statistical significance. CONCLUSION: Bacterial measures were related to inflammation and insulin resistance among diabetes-free adults. Twit Text FOAVip The subgingival microbiome, systemic inflammation and insulin resistance: The Oral Infections, Glucose Intolerance and Insulin Resistance Study ????J Clin Periodontol http://www.kidney.de/pget.php?&tw=1&pmid=27978598 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27978598 #FOAvip English Wikipedia <ref>{{Cite pmid|27978598 }}</ref> The subgingival microbiome, systemic inflammation and insulin resistance: The Oral Infections, Glucose Intolerance and Insulin Resistance Study #MMPMID27978598 Demmer RT ; Breskin A ; Rosenbaum M ; Zuk A ; LeDuc C ; Leibel R ; Paster B ; Desvarieux M ; Jacobs DR Jr ; Papapanou PN J Clin Periodontol 2017[Mar]; 44 (3 ): 255-265 PMID27978598 show ga BACKGROUND: Inflammation might link microbial exposures to insulin resistance. We investigated the cross-sectional association between periodontal microbiota, inflammation and insulin resistance. METHODS: The Oral Infections, Glucose Intolerance and Insulin Resistance Study (ORIGINS) enrolled 152 diabetes-free adults (77% female) aged 20-55 years (mean = 34 ± 10). Three hundred and four subgingival plaque samples were analysed using the Human Oral Microbe Identification Microarray to measure the relative abundances of 379 taxa. C-reactive protein, interleukin-6, tumour necrosis factor-? and adiponectin were assessed from venous blood and their z-scores were summed to create an inflammatory score (IS). Insulin resistance was defined via the HOMA-IR. Associations between the microbiota and both inflammation and HOMA-IR were explored using multivariable linear regressions; mediation analyses assessed the proportion of the association explained by inflammation. RESULTS: The IS was inversely associated with Actinobacteria and Proteobacteria and positively associated with Firmicutes and TM7 (p-values < 0.05). Proteobacteria levels were associated with insulin resistance (p < 0.05). Inflammation explained 30-98% of the observed associations between levels of Actinobacteria, Proteobacteria or Firmicutes and insulin resistance (p-values < 0.05). Eighteen individual taxa were associated with inflammation (p < 0.05) and 22 with insulin resistance (p < 0.05). No findings for individual taxa met Bonferroni-adjusted statistical significance. CONCLUSION: Bacterial measures were related to inflammation and insulin resistance among diabetes-free adults. |*Insulin Resistance [MESH] |*Microbiota [MESH] |Adult [MESH] |Cross-Sectional Studies [MESH] |Female [MESH] |Gingiva/*microbiology [MESH] |Glucose Intolerance [MESH] |Humans [MESH] |Inflammation/*microbiology [MESH] |Male [MESH] |Middle Aged [MESH] |Mouth Diseases/microbiology [MESH]The subgingival microbiome, systemic inflammation and insulin resistan(epmc).pdf DeepDyve Pubget Overpricing