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10.3389/fcimb.2017.00053 http://scihub22266oqcxt.onion/10.3389/fcimb.2017.00053 C5322174!5322174!28280716     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28280716&cmd=llinks): Failed to open stream: HTTP request failed! 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Aflatoxin B1 Induces Reactive Oxygen Species-Mediated Autophagy and Extracellular Trap Formation in Macrophages |pdf=|usr=}}{{28280716}} gab.com Text Aflatoxin B1 Induces Reactive Oxygen Species-Mediated Autophagy and Extracellular Trap Formation in Macrophages JO: Front Cell Infect Microbiol http://www.kidney.de/pget.php?&tw=1&pmid=28280716 #FOAvip Aflatoxins are a group of highly toxic mycotoxins with high carcinogenicity that are commonly found in foods. Aflatoxin B1 (AFB1) is the most toxic member of the aflatoxin family. A recent study reported that AFB1 can induce autophagy, but whether AFB1 can induce extracellular traps (ETs) and the relationships among innate immune responses, reactive oxygen species (ROS), and autophagy and the ETs induced by AFB1 remain unknown. Here, we demonstrated that AFB1 induced a complete autophagic process in macrophages (M?) (THP-1 cells and RAW264.7 cells). In addition, AFB1 induced the generation of M? ETs (METs) in a dose-dependent manner. In particular, the formation of METs significantly reduced the AFB1 content. Further analysis using specific inhibitors showed that the inhibition of either autophagy or ROS prevented MET formation caused by AFB1, indicating that autophagy and ROS were required for AFB1-induced MET formation. The inhibition of ROS prevented autophagy, indicating that ROS generation occurred upstream of AFB1-induced autophagy. Taken together, these data suggest that AFB1 induces ROS-mediated autophagy and ETs formation and an M1 phenotype in M?. Twit Text FOAVip Aflatoxin B1 Induces Reactive Oxygen Species-Mediated Autophagy and Extracellular Trap Formation in Macrophages ????Front Cell Infect Microbiol http://www.kidney.de/pget.php?&tw=1&pmid=28280716 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28280716 #FOAvip English Wikipedia <ref>{{Cite pmid|28280716}}</ref> Aflatoxin B1 Induces Reactive Oxygen Species-Mediated Autophagy and Extracellular Trap Formation in Macrophages #MMPMID28280716 An Y; Shi X; Tang X; Wang Y; Shen F; Zhang Q; Wang C; Jiang M; Liu M; Yu L Front Cell Infect Microbiol 2017[]; 7 (ä): ä PMID28280716show ga Aflatoxins are a group of highly toxic mycotoxins with high carcinogenicity that are commonly found in foods. Aflatoxin B1 (AFB1) is the most toxic member of the aflatoxin family. A recent study reported that AFB1 can induce autophagy, but whether AFB1 can induce extracellular traps (ETs) and the relationships among innate immune responses, reactive oxygen species (ROS), and autophagy and the ETs induced by AFB1 remain unknown. Here, we demonstrated that AFB1 induced a complete autophagic process in macrophages (M?) (THP-1 cells and RAW264.7 cells). In addition, AFB1 induced the generation of M? ETs (METs) in a dose-dependent manner. In particular, the formation of METs significantly reduced the AFB1 content. Further analysis using specific inhibitors showed that the inhibition of either autophagy or ROS prevented MET formation caused by AFB1, indicating that autophagy and ROS were required for AFB1-induced MET formation. The inhibition of ROS prevented autophagy, indicating that ROS generation occurred upstream of AFB1-induced autophagy. Taken together, these data suggest that AFB1 induces ROS-mediated autophagy and ETs formation and an M1 phenotype in M?. äAflatoxin B1 Induces Reactive Oxygen Species-Mediated Autophagy and Ex(epmc).pdf DeepDyve Pubget Overpricing