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10.1161/HYPERTENSIONAHA.116.07971 http://scihub22266oqcxt.onion/10.1161/HYPERTENSIONAHA.116.07971 C5142826!5142826!27698062     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27698062&cmd=llinks): Failed to open stream: HTTP request failed! 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Agonistic Autoantibodies to the Angiotensin II Type 1 Receptor Enhance ANG II Induced Renal Vascular Sensitivity and Reduce Renal Function during Pregnancy |pdf=|usr=}}{{27698062}} gab.com Text Agonistic Autoantibodies to the Angiotensin II Type 1 Receptor Enhance ANG II Induced Renal Vascular Sensitivity and Reduce Renal Function during Pregnancy JO: Hypertension http://www.kidney.de/pget.php?&tw=1&pmid=27698062 #FOAvip Preeclamptic women produce agonistic autoantibodies to the angiotensin II type 1 receptor (AT1-AA) and exhibit increased blood pressure (MAP), vascular sensitivity to angiotensin II (ANG II), and display a decrease in renal function. The objective of this study was to examine the renal hemodynamic changes during pregnancy in the presence of AT1-AAs with or without a slow pressor dose of ANGII. In this study MAP was elevated in all pregnant rats treated with ANG II with or without AT1-AA. Glomerular filtration rate (GFR) was reduced from 1.90 ±0.16 ml/min in normal pregnant (NP) to 1.20 ±0.08 in ANGII + AT1-AA rats. Renal blood flow (RBF) was decreased in ANGII + AT1-AA vs NP rats to 7.4 ±1.09 vs. 15.4 ±1.75 ml/min. Renal vascular resistance (RVR) was drastically increased between ANGII + AT1-AA vs NP rats (18.4 ±2.91 vs. 6.4 ±0.77 mmHg/ml/min). Isoprostane excretion was increased by a 3.5 fold in ANG II + AT1-AA vs. NP (1160±321 vs. 323±52 pg/ml). In conclusion ANG II and AT1-AA together, significantly decrease GFR by 37%, RBF by 50%, and caused a 3 fold increase in RVR and isoprostane levels vs NP rats. These data indicate the importance of AT1-AAs to enhance ANG II induced renal vasoconstriction and reduce renal function as mechanisms to cause hypertension as observed during preeclampsia. Twit Text FOAVip Agonistic Autoantibodies to the Angiotensin II Type 1 Receptor Enhance ANG II Induced Renal Vascular Sensitivity and Reduce Renal Function during Pregnancy ????Hypertension http://www.kidney.de/pget.php?&tw=1&pmid=27698062 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27698062 #FOAvip English Wikipedia <ref>{{Cite pmid|27698062}}</ref> Agonistic Autoantibodies to the Angiotensin II Type 1 Receptor Enhance ANG II Induced Renal Vascular Sensitivity and Reduce Renal Function during Pregnancy #MMPMID27698062 Cunningham MW; Williams JM; Amaral L; Usry N; Wallukat G; Dechend R; LaMarca B Hypertension 2016[Nov]; 68 (5): 1308-13 PMID27698062show ga Preeclamptic women produce agonistic autoantibodies to the angiotensin II type 1 receptor (AT1-AA) and exhibit increased blood pressure (MAP), vascular sensitivity to angiotensin II (ANG II), and display a decrease in renal function. The objective of this study was to examine the renal hemodynamic changes during pregnancy in the presence of AT1-AAs with or without a slow pressor dose of ANGII. In this study MAP was elevated in all pregnant rats treated with ANG II with or without AT1-AA. Glomerular filtration rate (GFR) was reduced from 1.90 ±0.16 ml/min in normal pregnant (NP) to 1.20 ±0.08 in ANGII + AT1-AA rats. Renal blood flow (RBF) was decreased in ANGII + AT1-AA vs NP rats to 7.4 ±1.09 vs. 15.4 ±1.75 ml/min. Renal vascular resistance (RVR) was drastically increased between ANGII + AT1-AA vs NP rats (18.4 ±2.91 vs. 6.4 ±0.77 mmHg/ml/min). Isoprostane excretion was increased by a 3.5 fold in ANG II + AT1-AA vs. NP (1160±321 vs. 323±52 pg/ml). In conclusion ANG II and AT1-AA together, significantly decrease GFR by 37%, RBF by 50%, and caused a 3 fold increase in RVR and isoprostane levels vs NP rats. These data indicate the importance of AT1-AAs to enhance ANG II induced renal vasoconstriction and reduce renal function as mechanisms to cause hypertension as observed during preeclampsia. äAgonistic Autoantibodies to the Angiotensin II Type 1 Receptor Enhance(epmc).pdf DeepDyve Pubget Overpricing