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10.1152/ajpheart.00948.2015 http://scihub22266oqcxt.onion/10.1152/ajpheart.00948.2015 C5142186!5142186!27496882     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Am+J+Physiol+Heart+Circ+Physiol 2016 ; 311 (3): H815-21 Nephropedia Template TP {{tp|p=27496882|t=2016. CXCL16 regulates renal injury and fibrosis in experimental renal artery stenosis |pdf=|usr=}}{{27496882}} gab.com Text CXCL16 regulates renal injury and fibrosis in experimental renal artery stenosis JO: Am J Physiol Heart Circ Physiol http://www.kidney.de/pget.php?&tw=1&pmid=27496882 #FOAvip Renal artery stenosis (RAS) is an important cause of chronic kidney disease. The underlying mechanisms are not well understood. Our study indicates that CXCL16 plays an important role in the pathogenesis of RAS-induced renal injury and fibrosis through regulation of myeloid fibroblast accumulation and inflammatory cell infiltration. Twit Text FOAVip CXCL16 regulates renal injury and fibrosis in experimental renal artery stenosis ????Am J Physiol Heart Circ Physiol http://www.kidney.de/pget.php?&tw=1&pmid=27496882 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27496882 #FOAvip English Wikipedia <ref>{{Cite pmid|27496882}}</ref> CXCL16 regulates renal injury and fibrosis in experimental renal artery stenosis #MMPMID27496882 Ma Z; Jin X; He L; Wang Y Am J Physiol Heart Circ Physiol 2016[Sep]; 311 (3): H815-21 PMID27496882show ga Renal artery stenosis (RAS) is an important cause of chronic kidney disease. The underlying mechanisms are not well understood. Our study indicates that CXCL16 plays an important role in the pathogenesis of RAS-induced renal injury and fibrosis through regulation of myeloid fibroblast accumulation and inflammatory cell infiltration. äCXCL16 regulates renal injury and fibrosis in experimental renal arter(epmc).pdf DeepDyve Pubget Overpricing