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10.7150/ijbs.16564 http://scihub22266oqcxt.onion/10.7150/ijbs.16564 C5118777!5118777!27877083     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Int+J+Biol+Sci 2016 ; 12 (11): 1309-18 Nephropedia Template TP {{tp|p=27877083|t=2016. Epstein-Barr virus lytic reactivation regulation and its pathogenic role in carcinogenesis |pdf=|usr=}}{{27877083}} gab.com Text Epstein-Barr virus lytic reactivation regulation and its pathogenic role in carcinogenesis JO: Int J Biol Sci http://www.kidney.de/pget.php?&tw=1&pmid=27877083 #FOAvip Epstein-Barr virus (EBV) has been associated with several types of human cancers. In the host, EBV can establish two alternative modes of life cycle, known as latent or lytic and the switch from latency to the lytic cycle is known as EBV reactivation. Although EBV in cancer cells is found mostly in latency, a small number of lytically-infected cells promote carcinogenesis through the release of growth factors and oncogenic cytokines. This review focuses on the mechanisms by which EBV reactivation is controlled by cellular and viral factors, and discusses how EBV lytic infection contributes to human malignancies. Twit Text FOAVip Epstein-Barr virus lytic reactivation regulation and its pathogenic role in carcinogenesis ????Int J Biol Sci http://www.kidney.de/pget.php?&tw=1&pmid=27877083 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27877083 #FOAvip English Wikipedia <ref>{{Cite pmid|27877083}}</ref> Epstein-Barr virus lytic reactivation regulation and its pathogenic role in carcinogenesis #MMPMID27877083 Li H; Liu S; Hu J; Luo X; Li N; M.Bode A; Cao Y Int J Biol Sci 2016[]; 12 (11): 1309-18 PMID27877083show ga Epstein-Barr virus (EBV) has been associated with several types of human cancers. In the host, EBV can establish two alternative modes of life cycle, known as latent or lytic and the switch from latency to the lytic cycle is known as EBV reactivation. Although EBV in cancer cells is found mostly in latency, a small number of lytically-infected cells promote carcinogenesis through the release of growth factors and oncogenic cytokines. This review focuses on the mechanisms by which EBV reactivation is controlled by cellular and viral factors, and discusses how EBV lytic infection contributes to human malignancies. äEpstein-Barr virus lytic reactivation regulation and its pathogenic ro(epmc).pdf DeepDyve Pubget Overpricing