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10.1128/JVI.01692-16 http://scihub22266oqcxt.onion/10.1128/JVI.01692-16 C5105643!5105643!27605675     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Virol 2016 ; 90 (22): 10050-3 Nephropedia Template TP {{tp|p=27605675|t=2016. Cell-Cell Sensing of Viral Infection by Plasmacytoid Dendritic Cells |pdf=|usr=}}{{27605675}} gab.com Text Cell-Cell Sensing of Viral Infection by Plasmacytoid Dendritic Cells JO: J Virol http://www.kidney.de/pget.php?&tw=1&pmid=27605675 #FOAvip All cells possess signaling pathways designed to trigger antiviral responses, notably characterized by type I interferon (IFN) production, upon recognition of invading viruses. Especially, host sensors recognize viral nucleic acids. Nonetheless, virtually all viruses have evolved potent strategies that preclude host responses within the infected cells. The plasmacytoid dendritic cell (pDC) is an immune cell type known as a robust type I IFN producer in response to viral infection. Evidence suggests that such functionality of the pDCs participates in viral clearance. Nonetheless, their contribution, which is likely complex and varies depending on the pathogen, is still enigmatic for many viruses. pDCs are not permissive to most viral infections, and consistently, recent examples suggest that pDCs respond to immunostimulatory viral RNA transferred via noninfectious and/or noncanonical viral/cellular carriers. Therefore, the pDC response likely bypasses innate signaling blockages induced by virus within infected cells. Importantly, the requirement for cell-cell contact is increasingly recognized as a hallmark of the pDC-mediated antiviral state, triggered by evolutionarily divergent RNA viruses. Twit Text FOAVip Cell-Cell Sensing of Viral Infection by Plasmacytoid Dendritic Cells ????J Virol http://www.kidney.de/pget.php?&tw=1&pmid=27605675 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27605675 #FOAvip English Wikipedia <ref>{{Cite pmid|27605675}}</ref> Cell-Cell Sensing of Viral Infection by Plasmacytoid Dendritic Cells #MMPMID27605675 Webster B; Assil S; Dreux M J Virol 2016[Nov]; 90 (22): 10050-3 PMID27605675show ga All cells possess signaling pathways designed to trigger antiviral responses, notably characterized by type I interferon (IFN) production, upon recognition of invading viruses. Especially, host sensors recognize viral nucleic acids. Nonetheless, virtually all viruses have evolved potent strategies that preclude host responses within the infected cells. The plasmacytoid dendritic cell (pDC) is an immune cell type known as a robust type I IFN producer in response to viral infection. Evidence suggests that such functionality of the pDCs participates in viral clearance. Nonetheless, their contribution, which is likely complex and varies depending on the pathogen, is still enigmatic for many viruses. pDCs are not permissive to most viral infections, and consistently, recent examples suggest that pDCs respond to immunostimulatory viral RNA transferred via noninfectious and/or noncanonical viral/cellular carriers. Therefore, the pDC response likely bypasses innate signaling blockages induced by virus within infected cells. Importantly, the requirement for cell-cell contact is increasingly recognized as a hallmark of the pDC-mediated antiviral state, triggered by evolutionarily divergent RNA viruses. äCell-Cell Sensing of Viral Infection by Plasmacytoid Dendritic Cells (epmc).pdf DeepDyve Pubget Overpricing