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10.1007/s00467-016-3394-5 http://scihub22266oqcxt.onion/10.1007/s00467-016-3394-5 C5099107!5099107!27155873     free     free     free Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Pediatr+Nephrol 2017 ; 32 (4): 577-87 Nephropedia Template TP {{tp|p=27155873|t=2017. Tubulointerstitial Nephritis: Diagnosis, Treatment and Monitoring |pdf=|usr=}}{{27155873}} gab.com Text Tubulointerstitial Nephritis: Diagnosis, Treatment and Monitoring JO: Pediatr Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=27155873 #FOAvip Tubulointerstitial nephritis (TIN) is a frequent cause of acute kidney injury (AKI) that can lead to chronic kidney disease (CKD). TIN is associated with an immune-mediated infiltration of the kidney interstitium by inflammatory cells, which may progress to fibrosis. Patients often present with non-specific symptoms, which can lead to delayed diagnosis and treatment of the disease. Etiology of TIN can be drug-induced, infectious, idiopathic, genetic, or related to a systemic inflammatory condition such as tubulointerstitial nephritis and uveitis (TINU) syndrome, inflammatory bowel disease, or IgG4-associated immune complex multiorgan autoimmune disease (MAD). It is imperative to have a high clinical suspicion for TIN in order to remove potential offending agents and treat any associated systemic diseases. Treatment is ultimately dependent on underlying etiology. While there are no randomized controlled clinical trials to assess treatment choice and efficacy in TIN, corticosteroids have been a mainstay of therapy and recent studies have suggested a possible role for mycophenolate mofetil. Urinary biomarkers such as alpha1-microglobulin and beta2-microglobulin may help diagnose and monitor disease activity in TIN. Screening for TIN should be implemented in children with inflammatory bowel disease, uveitis, or IgG4-associated MAD. Twit Text FOAVip Tubulointerstitial Nephritis: Diagnosis, Treatment and Monitoring ????Pediatr Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=27155873 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27155873 #FOAvip English Wikipedia <ref>{{Cite pmid|27155873}}</ref> Tubulointerstitial Nephritis: Diagnosis, Treatment and Monitoring #MMPMID27155873 Joyce E; Glasner P; Ranganathan S; Swiatecka-Urban A Pediatr Nephrol 2017[Apr]; 32 (4): 577-87 PMID27155873show ga Tubulointerstitial nephritis (TIN) is a frequent cause of acute kidney injury (AKI) that can lead to chronic kidney disease (CKD). TIN is associated with an immune-mediated infiltration of the kidney interstitium by inflammatory cells, which may progress to fibrosis. Patients often present with non-specific symptoms, which can lead to delayed diagnosis and treatment of the disease. Etiology of TIN can be drug-induced, infectious, idiopathic, genetic, or related to a systemic inflammatory condition such as tubulointerstitial nephritis and uveitis (TINU) syndrome, inflammatory bowel disease, or IgG4-associated immune complex multiorgan autoimmune disease (MAD). It is imperative to have a high clinical suspicion for TIN in order to remove potential offending agents and treat any associated systemic diseases. Treatment is ultimately dependent on underlying etiology. While there are no randomized controlled clinical trials to assess treatment choice and efficacy in TIN, corticosteroids have been a mainstay of therapy and recent studies have suggested a possible role for mycophenolate mofetil. Urinary biomarkers such as alpha1-microglobulin and beta2-microglobulin may help diagnose and monitor disease activity in TIN. Screening for TIN should be implemented in children with inflammatory bowel disease, uveitis, or IgG4-associated MAD. äTubulointerstitial Nephritis: Diagnosis, Treatment and Monitoring (epmc).pdf DeepDyve Pubget Overpricing