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10.1042/BST20160146 http://scihub22266oqcxt.onion/10.1042/BST20160146 C5095905!5095905!27911713     free     free     free Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Biochem+Soc+Trans 2016 ; 44 (5): 1305-12 Nephropedia Template TP {{tp|p=27911713|t=2016. Regulatory mechanisms of phosphatase of regenerating liver (PRL)-3 |pdf=|usr=}}{{27911713}} gab.com Text Regulatory mechanisms of phosphatase of regenerating liver (PRL)-3 JO: Biochem Soc Trans http://www.kidney.de/pget.php?&tw=1&pmid=27911713 #FOAvip The phosphatase of regenerating liver (PRL)-3 is overexpressed in many human cancer types and tumor metastases when compared with healthy tissues. Different pathways and mechanisms have been suggested to modulate PRL-3 expression levels and activity, giving some valuable insights but still leaving an incomplete picture. Investigating these mechanisms could provide new targets for therapeutic drug development. Here, we present an updated overview and summarize recent findings concerning the different PRL-3 expression regulatory mechanisms and posttranslational modifications suggested to modulate the activity, localization, or stability of this phosphatase. Twit Text FOAVip Regulatory mechanisms of phosphatase of regenerating liver (PRL)-3 ????Biochem Soc Trans http://www.kidney.de/pget.php?&tw=1&pmid=27911713 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27911713 #FOAvip English Wikipedia <ref>{{Cite pmid|27911713}}</ref> Regulatory mechanisms of phosphatase of regenerating liver (PRL)-3 #MMPMID27911713 Rubio T; Köhn M Biochem Soc Trans 2016[Oct]; 44 (5): 1305-12 PMID27911713show ga The phosphatase of regenerating liver (PRL)-3 is overexpressed in many human cancer types and tumor metastases when compared with healthy tissues. Different pathways and mechanisms have been suggested to modulate PRL-3 expression levels and activity, giving some valuable insights but still leaving an incomplete picture. Investigating these mechanisms could provide new targets for therapeutic drug development. Here, we present an updated overview and summarize recent findings concerning the different PRL-3 expression regulatory mechanisms and posttranslational modifications suggested to modulate the activity, localization, or stability of this phosphatase. äRegulatory mechanisms of phosphatase of regenerating liver (PRL)-3 (epmc).pdf DeepDyve Pubget Overpricing