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10.5009/gnl15414 http://scihub22266oqcxt.onion/10.5009/gnl15414 C5087926!5087926!27784845     free     free     free Deprecated: Implicit conversion from float 215.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Gut+Liver 2016 ; 10 (6): 881-9 Nephropedia Template TP {{tp|p=27784845|t=2016. Potassium Channelopathies and Gastrointestinal Ulceration |pdf=|usr=}}{{27784845}} gab.com Text Potassium Channelopathies and Gastrointestinal Ulceration JO: Gut Liver http://www.kidney.de/pget.php?&tw=1&pmid=27784845 #FOAvip Potassium channels and transporters maintain potassium homeostasis and play significant roles in several different biological actions via potassium ion regulation. In previous decades, the key revelations that potassium channels and transporters are involved in the production of gastric acid and the regulation of secretion in the stomach have been recognized. Drugs used to treat peptic ulceration are often potassium transporter inhibitors. It has also been reported that potassium channels are involved in ulcerative colitis. Direct toxicity to the intestines from nonsteroidal anti-inflammatory drugs has been associated with altered potassium channel activities. Several reports have indicated that the long-term use of the antianginal drug Nicorandil, an adenosine triphosphate-sensitive potassium channel opener, increases the chances of ulceration and perforation from the oral to anal regions throughout the gastrointestinal (GI) tract. Several of these drug features provide further insights into the role of potassium channels in the occurrence of ulceration in the GI tract. The purpose of this review is to investigate whether potassium channelopathies are involved in the mechanisms responsible for ulceration that occurs throughout the GI tract. Twit Text FOAVip Potassium Channelopathies and Gastrointestinal Ulceration ????Gut Liver http://www.kidney.de/pget.php?&tw=1&pmid=27784845 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27784845 #FOAvip English Wikipedia <ref>{{Cite pmid|27784845}}</ref> Potassium Channelopathies and Gastrointestinal Ulceration #MMPMID27784845 Han J; Lee SH; Giebisch G; Wang T Gut Liver 2016[Nov]; 10 (6): 881-9 PMID27784845show ga Potassium channels and transporters maintain potassium homeostasis and play significant roles in several different biological actions via potassium ion regulation. In previous decades, the key revelations that potassium channels and transporters are involved in the production of gastric acid and the regulation of secretion in the stomach have been recognized. Drugs used to treat peptic ulceration are often potassium transporter inhibitors. It has also been reported that potassium channels are involved in ulcerative colitis. Direct toxicity to the intestines from nonsteroidal anti-inflammatory drugs has been associated with altered potassium channel activities. Several reports have indicated that the long-term use of the antianginal drug Nicorandil, an adenosine triphosphate-sensitive potassium channel opener, increases the chances of ulceration and perforation from the oral to anal regions throughout the gastrointestinal (GI) tract. Several of these drug features provide further insights into the role of potassium channels in the occurrence of ulceration in the GI tract. The purpose of this review is to investigate whether potassium channelopathies are involved in the mechanisms responsible for ulceration that occurs throughout the GI tract. äPotassium Channelopathies and Gastrointestinal Ulceration (epmc).pdf DeepDyve Pubget Overpricing