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10.3389/fphys.2016.00482 http://scihub22266oqcxt.onion/10.3389/fphys.2016.00482 C5081373!5081373!27833564     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Front+Physiol 2016 ; 7 (ä): ä Nephropedia Template TP {{tp|p=27833564|t=2016. The Bohr Effect Is Not a Likely Promoter of Renal Preglomerular Oxygen Shunting |pdf=|usr=}}{{27833564}} gab.com Text The Bohr Effect Is Not a Likely Promoter of Renal Preglomerular Oxygen Shunting JO: Front Physiol http://www.kidney.de/pget.php?&tw=1&pmid=27833564 #FOAvip The aim of this study was to evaluate whether possible preglomerular arterial-to-venous oxygen shunting is affected by the interaction between renal preglomerular carbon dioxide and oxygen transport. We hypothesized that a reverse (venous-to-arterial) shunting of carbon dioxide will increase partial pressure of carbon dioxide and decrease pH in the arteries and thereby lead to increased oxygen offloading and consequent oxygen shunting. To test this hypothesis, we employed a segment-wise three-dimensional computational model of coupled renal oxygen and carbon dioxide transport, wherein coupling is achieved by shifting the oxygen-hemoglobin dissociation curve in dependence of local changes in partial pressure of carbon dioxide and pH. The model suggests that primarily due to the high buffering capacity of blood, there is only marginally increased acidity in the preglomerular vasculature compared to systemic arterial blood caused by carbon dioxide shunting. Furthermore, effects of carbon dioxide transport do not promote but rather impair preglomerular oxygen shunting, as the increase in acidity is higher in the veins compared to that in the arteries. We conclude that while substantial arterial-to-venous oxygen shunting might take place in the postglomerular vasculature, the net amount of oxygen shunted at the preglomerular vasculature appears to be marginal. Twit Text FOAVip The Bohr Effect Is Not a Likely Promoter of Renal Preglomerular Oxygen Shunting ????Front Physiol http://www.kidney.de/pget.php?&tw=1&pmid=27833564 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27833564 #FOAvip English Wikipedia <ref>{{Cite pmid|27833564}}</ref> The Bohr Effect Is Not a Likely Promoter of Renal Preglomerular Oxygen Shunting #MMPMID27833564 Olgac U; Kurtcuoglu V Front Physiol 2016[]; 7 (ä): ä PMID27833564show ga The aim of this study was to evaluate whether possible preglomerular arterial-to-venous oxygen shunting is affected by the interaction between renal preglomerular carbon dioxide and oxygen transport. We hypothesized that a reverse (venous-to-arterial) shunting of carbon dioxide will increase partial pressure of carbon dioxide and decrease pH in the arteries and thereby lead to increased oxygen offloading and consequent oxygen shunting. To test this hypothesis, we employed a segment-wise three-dimensional computational model of coupled renal oxygen and carbon dioxide transport, wherein coupling is achieved by shifting the oxygen-hemoglobin dissociation curve in dependence of local changes in partial pressure of carbon dioxide and pH. The model suggests that primarily due to the high buffering capacity of blood, there is only marginally increased acidity in the preglomerular vasculature compared to systemic arterial blood caused by carbon dioxide shunting. Furthermore, effects of carbon dioxide transport do not promote but rather impair preglomerular oxygen shunting, as the increase in acidity is higher in the veins compared to that in the arteries. We conclude that while substantial arterial-to-venous oxygen shunting might take place in the postglomerular vasculature, the net amount of oxygen shunted at the preglomerular vasculature appears to be marginal. äThe Bohr Effect Is Not a Likely Promoter of Renal Preglomerular Oxygen(epmc).pdf DeepDyve Pubget Overpricing