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10.1007/s11906-016-0635-8 http://scihub22266oqcxt.onion/10.1007/s11906-016-0635-8 C5078991!5078991!26957306     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Curr+Hypertens+Rep 2016 ; 18 (4): 30 Nephropedia Template TP {{tp|p=26957306|t=2016. Neural Control of Non-vasomotor Organs in Hypertension |pdf=|usr=}}{{26957306}} gab.com Text Neural Control of Non-vasomotor Organs in Hypertension JO: Curr Hypertens Rep http://www.kidney.de/pget.php?&tw=1&pmid=26957306 #FOAvip Hypertension affects over 25 % of the population with the incidence continuing to rise, due in part to the growing obesity epidemic. Chronic elevations in sympathetic nerve activity (SNA) are a hallmark of the disease and contribute to elevations in blood pressure through influences on the vasculature, kidney, and heart (i.e., neurogenic hypertension). In this regard, a number of central nervous system mechanisms and neural pathways have emerged as crucial in chronically elevating SNA. However, it is important to consider that ?sympathetic signatures? are present, with differential increases in SNA to regional organs that are dependent upon the disease progression. Here, we discuss recent findings on the central nervous system mechanisms and autonomic regulatory networks involved in neurogenic hypertension, in both non-obesity- and obesity-associated hypertension, with an emphasis on angiotensin-II, salt, oxidative and endoplasmic reticulum stress, inflammation, and the adipokine leptin. Twit Text FOAVip Neural Control of Non-vasomotor Organs in Hypertension ????Curr Hypertens Rep http://www.kidney.de/pget.php?&tw=1&pmid=26957306 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26957306 #FOAvip English Wikipedia <ref>{{Cite pmid|26957306}}</ref> Neural Control of Non-vasomotor Organs in Hypertension #MMPMID26957306 Hurr C; Young CN Curr Hypertens Rep 2016[Apr]; 18 (4): 30 PMID26957306show ga Hypertension affects over 25 % of the population with the incidence continuing to rise, due in part to the growing obesity epidemic. Chronic elevations in sympathetic nerve activity (SNA) are a hallmark of the disease and contribute to elevations in blood pressure through influences on the vasculature, kidney, and heart (i.e., neurogenic hypertension). In this regard, a number of central nervous system mechanisms and neural pathways have emerged as crucial in chronically elevating SNA. However, it is important to consider that ?sympathetic signatures? are present, with differential increases in SNA to regional organs that are dependent upon the disease progression. Here, we discuss recent findings on the central nervous system mechanisms and autonomic regulatory networks involved in neurogenic hypertension, in both non-obesity- and obesity-associated hypertension, with an emphasis on angiotensin-II, salt, oxidative and endoplasmic reticulum stress, inflammation, and the adipokine leptin. äNeural Control of Non-vasomotor Organs in Hypertension (epmc).pdf DeepDyve Pubget Overpricing