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10.1080/23723556.2015.1078923 http://scihub22266oqcxt.onion/10.1080/23723556.2015.1078923 C5068182!5068182!27857968     free     free     free Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Mol+Cell+Oncol 2016 ; 3 (5): ä Nephropedia Template TP {{tp|p=27857968|t=2016. Tor-dependent post-transcriptional regulation of autophagy: Implications for cancer therapeutics |pdf=|usr=}}{{27857968}} gab.com Text Tor-dependent post-transcriptional regulation of autophagy: Implications for cancer therapeutics JO: Mol Cell Oncol http://www.kidney.de/pget.php?&tw=1&pmid=27857968 #FOAvip Paradoxically, both anticancer immunosurveillance and tumor progression have been associated with intact autophagy, which is regulated by the target of rapamycin (Tor1). Here, we describe the potential impact on the design of cancer therapeutics of a newly described highly conserved post-transcriptional mechanism whereby Tor regulates autophagy. Twit Text FOAVip Tor-dependent post-transcriptional regulation of autophagy: Implications for cancer therapeutics ????Mol Cell Oncol http://www.kidney.de/pget.php?&tw=1&pmid=27857968 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27857968 #FOAvip English Wikipedia <ref>{{Cite pmid|27857968}}</ref> Tor-dependent post-transcriptional regulation of autophagy: Implications for cancer therapeutics #MMPMID27857968 Hu G; McQuiston T; Bernard A; Park YD; Qiu J; Vural A; Zhang N; Waterman SR; Blewett NH; Myers TG; Kehrl JH; Uzel G; Klionsky DJ; Williamson PR Mol Cell Oncol 2016[]; 3 (5): ä PMID27857968show ga Paradoxically, both anticancer immunosurveillance and tumor progression have been associated with intact autophagy, which is regulated by the target of rapamycin (Tor1). Here, we describe the potential impact on the design of cancer therapeutics of a newly described highly conserved post-transcriptional mechanism whereby Tor regulates autophagy. äTor-dependent post-transcriptional regulation of autophagy: Implicatio(epmc).pdf DeepDyve Pubget Overpricing