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10.1155/2016/6969040 http://scihub22266oqcxt.onion/10.1155/2016/6969040 C5061963!5061963!27777961     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Diabetes+Res 2016 ; 2016 (ä): ä Nephropedia Template TP {{tp|p=27777961|t=2016. Sweet Bones: The Pathogenesis of Bone Alteration in Diabetes |pdf=|usr=}}{{27777961}} gab.com Text Sweet Bones: The Pathogenesis of Bone Alteration in Diabetes JO: J Diabetes Res http://www.kidney.de/pget.php?&tw=1&pmid=27777961 #FOAvip Diabetic patients have increased fracture risk. The pathogenesis underlying the status of bone alterations in diabetes mellitus is not completely understood but is multifactorial. The major deficits appear to be related to a deficit in mineralized surface area, a decrement in the rate of mineral apposition, deceased osteoid surface, depressed osteoblast activity, and decreased numbers of osteoclasts due to abnormal insulin signaling pathway. Other prominent features of diabetes mellitus are an increased urinary excretion of calcium and magnesium, accumulation of advanced glycation end products, and oxidative stress leading to sweet bones (altered bone's strength, metabolism, and structure). Every diabetic patient should be assessed for risk factors for fractures and osteoporosis. The pathogenesis of the bone alterations in diabetes mellitus as well as their molecular mechanisms needs further study. Twit Text FOAVip Sweet Bones: The Pathogenesis of Bone Alteration in Diabetes ????J Diabetes Res http://www.kidney.de/pget.php?&tw=1&pmid=27777961 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27777961 #FOAvip English Wikipedia <ref>{{Cite pmid|27777961}}</ref> Sweet Bones: The Pathogenesis of Bone Alteration in Diabetes #MMPMID27777961 Al-Hariri M J Diabetes Res 2016[]; 2016 (ä): ä PMID27777961show ga Diabetic patients have increased fracture risk. The pathogenesis underlying the status of bone alterations in diabetes mellitus is not completely understood but is multifactorial. The major deficits appear to be related to a deficit in mineralized surface area, a decrement in the rate of mineral apposition, deceased osteoid surface, depressed osteoblast activity, and decreased numbers of osteoclasts due to abnormal insulin signaling pathway. Other prominent features of diabetes mellitus are an increased urinary excretion of calcium and magnesium, accumulation of advanced glycation end products, and oxidative stress leading to sweet bones (altered bone's strength, metabolism, and structure). Every diabetic patient should be assessed for risk factors for fractures and osteoporosis. The pathogenesis of the bone alterations in diabetes mellitus as well as their molecular mechanisms needs further study. äSweet Bones: The Pathogenesis of Bone Alteration in Diabetes (epmc).pdf DeepDyve Pubget Overpricing