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10.1155/2016/4576012 http://scihub22266oqcxt.onion/10.1155/2016/4576012 C5050358!5050358 !27738642     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27738642 &cmd=llinks): Failed to open stream: HTTP request failed! 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Blood-Brain Barrier Disruption Induced by Chronic Sleep Loss: Low-Grade Inflammation May Be the Link |pdf=|usr=}}{{27738642 }} gab.com Text Blood-Brain Barrier Disruption Induced by Chronic Sleep Loss: Low-Grade Inflammation May Be the Link JO: J Immunol Res http://www.kidney.de/pget.php?&tw=1&pmid=27738642 #FOAvip Sleep is a vital phenomenon related to immunomodulation at the central and peripheral level. Sleep deficient in duration and/or quality is a common problem in the modern society and is considered a risk factor to develop neurodegenerative diseases. Sleep loss in rodents induces blood-brain barrier disruption and the underlying mechanism is still unknown. Several reports indicate that sleep loss induces a systemic low-grade inflammation characterized by the release of several molecules, such as cytokines, chemokines, and acute-phase proteins; all of them may promote changes in cellular components of the blood-brain barrier, particularly on brain endothelial cells. In the present review we discuss the role of inflammatory mediators that increase during sleep loss and their association with general disturbances in peripheral endothelium and epithelium and how those inflammatory mediators may alter the blood-brain barrier. Finally, this manuscript proposes a hypothetical mechanism by which sleep loss may induce blood-brain barrier disruption, emphasizing the regulatory effect of inflammatory molecules on tight junction proteins. Twit Text FOAVip Blood-Brain Barrier Disruption Induced by Chronic Sleep Loss: Low-Grade Inflammation May Be the Link ????J Immunol Res http://www.kidney.de/pget.php?&tw=1&pmid=27738642 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27738642 #FOAvip English Wikipedia <ref>{{Cite pmid|27738642 }}</ref> Blood-Brain Barrier Disruption Induced by Chronic Sleep Loss: Low-Grade Inflammation May Be the Link #MMPMID27738642 Hurtado-Alvarado G ; Domínguez-Salazar E ; Pavon L ; Velázquez-Moctezuma J ; Gómez-González B J Immunol Res 2016[]; 2016 (ä): 4576012 PMID27738642 show ga Sleep is a vital phenomenon related to immunomodulation at the central and peripheral level. Sleep deficient in duration and/or quality is a common problem in the modern society and is considered a risk factor to develop neurodegenerative diseases. Sleep loss in rodents induces blood-brain barrier disruption and the underlying mechanism is still unknown. Several reports indicate that sleep loss induces a systemic low-grade inflammation characterized by the release of several molecules, such as cytokines, chemokines, and acute-phase proteins; all of them may promote changes in cellular components of the blood-brain barrier, particularly on brain endothelial cells. In the present review we discuss the role of inflammatory mediators that increase during sleep loss and their association with general disturbances in peripheral endothelium and epithelium and how those inflammatory mediators may alter the blood-brain barrier. Finally, this manuscript proposes a hypothetical mechanism by which sleep loss may induce blood-brain barrier disruption, emphasizing the regulatory effect of inflammatory molecules on tight junction proteins. |Acute-Phase Proteins/metabolism [MESH] |Animals [MESH] |Blood-Brain Barrier/immunology/*physiopathology [MESH] |Brain/*physiopathology [MESH] |Cytokines/physiology [MESH] |Endothelial Cells/physiology [MESH] |Epithelial Cells/physiology [MESH] |Gastrointestinal Microbiome/physiology [MESH] |Humans [MESH] |Inflammation Mediators/*physiology [MESH] |Inflammation/*immunology/physiopathology [MESH] |Neurodegenerative Diseases/etiology/physiopathology [MESH] |Rats [MESH]Blood-Brain Barrier Disruption Induced by Chronic Sleep Loss: Low-Grad(epmc).pdf DeepDyve Pubget Overpricing