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10.3390/ijms17091575 http://scihub22266oqcxt.onion/10.3390/ijms17091575 C5037841!5037841!27657051     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27657051&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Int+J+Mol+Sci 2016 ; 17 (9): ä Nephropedia Template TP {{tp|p=27657051|t=2016. Molecular Pathogenesis of NASH |pdf=|usr=}}{{27657051}} gab.com Text Molecular Pathogenesis of NASH JO: Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=27657051 #FOAvip Nonalcoholic steatohepatitis (NASH) is the main cause of chronic liver disease in the Western world and a major health problem, owing to its close association with obesity, diabetes, and the metabolic syndrome. NASH progression results from numerous events originating within the liver, as well as from signals derived from the adipose tissue and the gastrointestinal tract. In a fraction of NASH patients, disease may progress, eventually leading to advanced fibrosis, cirrhosis and hepatocellular carcinoma. Understanding the mechanisms leading to NASH and its evolution to cirrhosis is critical to identifying effective approaches for the treatment of this condition. In this review, we focus on some of the most recent data reported on the pathogenesis of NASH and its fibrogenic progression, highlighting potential targets for treatment or identification of biomarkers of disease progression. Twit Text FOAVip Molecular Pathogenesis of NASH ????Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=27657051 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27657051 #FOAvip English Wikipedia <ref>{{Cite pmid|27657051}}</ref> Molecular Pathogenesis of NASH #MMPMID27657051 Caligiuri A; Gentilini A; Marra F Int J Mol Sci 2016[Sep]; 17 (9): ä PMID27657051show ga Nonalcoholic steatohepatitis (NASH) is the main cause of chronic liver disease in the Western world and a major health problem, owing to its close association with obesity, diabetes, and the metabolic syndrome. NASH progression results from numerous events originating within the liver, as well as from signals derived from the adipose tissue and the gastrointestinal tract. In a fraction of NASH patients, disease may progress, eventually leading to advanced fibrosis, cirrhosis and hepatocellular carcinoma. Understanding the mechanisms leading to NASH and its evolution to cirrhosis is critical to identifying effective approaches for the treatment of this condition. In this review, we focus on some of the most recent data reported on the pathogenesis of NASH and its fibrogenic progression, highlighting potential targets for treatment or identification of biomarkers of disease progression. äMolecular Pathogenesis of NASH (epmc).pdf DeepDyve Pubget Overpricing