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10.3390/ijms17091511 http://scihub22266oqcxt.onion/10.3390/ijms17091511 C5037788!5037788!27618031     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Int+J+Mol+Sci 2016 ; 17 (9): ä Nephropedia Template TP {{tp|p=27618031|t=2016. Molecular Imaging of Vulnerable Atherosclerotic Plaques in Animal Models |pdf=|usr=}}{{27618031}} gab.com Text Molecular Imaging of Vulnerable Atherosclerotic Plaques in Animal Models JO: Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=27618031 #FOAvip Atherosclerosis is characterized by intimal plaques of the arterial vessels that develop slowly and, in some cases, may undergo spontaneous rupture with subsequent heart attack or stroke. Currently, noninvasive diagnostic tools are inadequate to screen atherosclerotic lesions at high risk of acute complications. Therefore, the attention of the scientific community has been focused on the use of molecular imaging for identifying vulnerable plaques. Genetically engineered murine models such as ApoE?/? and ApoE?/?Fbn1C1039G+/? mice have been shown to be useful for testing new probes targeting biomarkers of relevant molecular processes for the characterization of vulnerable plaques, such as vascular endothelial growth factor receptor (VEGFR)-1, VEGFR-2, intercellular adhesion molecule (ICAM)-1, P-selectin, and integrins, and for the potential development of translational tools to identify high-risk patients who could benefit from early therapeutic interventions. This review summarizes the main animal models of vulnerable plaques, with an emphasis on genetically altered mice, and the state-of-the-art preclinical molecular imaging strategies. Twit Text FOAVip Molecular Imaging of Vulnerable Atherosclerotic Plaques in Animal Models ????Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=27618031 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27618031 #FOAvip English Wikipedia <ref>{{Cite pmid|27618031}}</ref> Molecular Imaging of Vulnerable Atherosclerotic Plaques in Animal Models #MMPMID27618031 Gargiulo S; Gramanzini M; Mancini M Int J Mol Sci 2016[Sep]; 17 (9): ä PMID27618031show ga Atherosclerosis is characterized by intimal plaques of the arterial vessels that develop slowly and, in some cases, may undergo spontaneous rupture with subsequent heart attack or stroke. Currently, noninvasive diagnostic tools are inadequate to screen atherosclerotic lesions at high risk of acute complications. Therefore, the attention of the scientific community has been focused on the use of molecular imaging for identifying vulnerable plaques. Genetically engineered murine models such as ApoE?/? and ApoE?/?Fbn1C1039G+/? mice have been shown to be useful for testing new probes targeting biomarkers of relevant molecular processes for the characterization of vulnerable plaques, such as vascular endothelial growth factor receptor (VEGFR)-1, VEGFR-2, intercellular adhesion molecule (ICAM)-1, P-selectin, and integrins, and for the potential development of translational tools to identify high-risk patients who could benefit from early therapeutic interventions. This review summarizes the main animal models of vulnerable plaques, with an emphasis on genetically altered mice, and the state-of-the-art preclinical molecular imaging strategies. äMolecular Imaging of Vulnerable Atherosclerotic Plaques in Animal Mode(epmc).pdf DeepDyve Pubget Overpricing