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10.1590/1414-431X20165431 http://scihub22266oqcxt.onion/10.1590/1414-431X20165431 C5018691!5018691 !27599201     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27599201 &cmd=llinks): Failed to open stream: HTTP request failed! 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Angiotensin-(1-7) inhibits inflammation and oxidative stress to relieve lung injury induced by chronic intermittent hypoxia in rats |pdf=|usr=}}{{27599201 }} gab.com Text Angiotensin-(1-7) inhibits inflammation and oxidative stress to relieve lung injury induced by chronic intermittent hypoxia in rats JO: Braz J Med Biol Res http://www.kidney.de/pget.php?&tw=1&pmid=27599201 #FOAvip Obstructive sleep apnea is associated with inflammation and oxidative stress in lung tissues and can lead to metabolic abnormalities. We investigated the effects of angiotensin1-7 [Ang-(1-7)] on lung injury in rats induced by chronic intermittent hypoxia (CIH). We randomly assigned 32 male Sprague-Dawley rats (180-200 g) to normoxia control (NC), CIH-untreated (uCIH), Ang-(1-7)-treated normoxia control (N-A), and Ang-(1-7)-treated CIH (CIH-A) groups. Oxidative stress biomarkers were measured in lung tissues, and expression of NADPH oxidase 4 (Nox4) and Nox subunits (p22phox, and p47phox) was determined by Western blot and reverse transcription-polymerase chain reaction. Pulmonary pathological changes were more evident in the uCIH group than in the other groups. Enzyme-linked immunosorbent assays and immunohistochemical staining showed that inflammatory factor concentrations in serum and lung tissues in the uCIH group were significantly higher than those in the NC and N-A groups. Expression of inflammatory factors was significantly higher in the CIH-A group than in the NC and N-A groups, but was lower than in the uCIH group (P<0.01). Oxidative stress was markedly higher in the uCIH group than in the NC and N-A groups. Expression of Nox4 and its subunits was also increased in the uCIH group. These changes were attenuated upon Ang-(1-7) treatment. In summary, treatment with Ang-(1-7) reversed signs of CIH-induced lung injury via inhibition of inflammation and oxidative stress. Twit Text FOAVip Angiotensin-(1-7) inhibits inflammation and oxidative stress to relieve lung injury induced by chronic intermittent hypoxia in rats ????Braz J Med Biol Res http://www.kidney.de/pget.php?&tw=1&pmid=27599201 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27599201 #FOAvip English Wikipedia <ref>{{Cite pmid|27599201 }}</ref> Angiotensin-(1-7) inhibits inflammation and oxidative stress to relieve lung injury induced by chronic intermittent hypoxia in rats #MMPMID27599201 Lu W ; Kang J ; Hu K ; Tang S ; Zhou X ; Yu S ; Li Y ; Xu L Braz J Med Biol Res 2016[Sep]; 49 (10 ): e5431 PMID27599201 show ga Obstructive sleep apnea is associated with inflammation and oxidative stress in lung tissues and can lead to metabolic abnormalities. We investigated the effects of angiotensin1-7 [Ang-(1-7)] on lung injury in rats induced by chronic intermittent hypoxia (CIH). We randomly assigned 32 male Sprague-Dawley rats (180-200 g) to normoxia control (NC), CIH-untreated (uCIH), Ang-(1-7)-treated normoxia control (N-A), and Ang-(1-7)-treated CIH (CIH-A) groups. Oxidative stress biomarkers were measured in lung tissues, and expression of NADPH oxidase 4 (Nox4) and Nox subunits (p22phox, and p47phox) was determined by Western blot and reverse transcription-polymerase chain reaction. Pulmonary pathological changes were more evident in the uCIH group than in the other groups. Enzyme-linked immunosorbent assays and immunohistochemical staining showed that inflammatory factor concentrations in serum and lung tissues in the uCIH group were significantly higher than those in the NC and N-A groups. Expression of inflammatory factors was significantly higher in the CIH-A group than in the NC and N-A groups, but was lower than in the uCIH group (P<0.01). Oxidative stress was markedly higher in the uCIH group than in the NC and N-A groups. Expression of Nox4 and its subunits was also increased in the uCIH group. These changes were attenuated upon Ang-(1-7) treatment. In summary, treatment with Ang-(1-7) reversed signs of CIH-induced lung injury via inhibition of inflammation and oxidative stress. |Angiotensin I/*pharmacology [MESH] |Animals [MESH] |Blotting, Western [MESH] |Cytokines/analysis [MESH] |Enzyme-Linked Immunosorbent Assay [MESH] |Hypoxia/*complications [MESH] |Immunohistochemistry [MESH] |Inflammation/*drug therapy/pathology [MESH] |Lung Injury/*drug therapy/*etiology/metabolism [MESH] |Lung/drug effects/pathology [MESH] |Male [MESH] |Malondialdehyde/analysis [MESH] |Oxidative Stress/*drug effects [MESH] |Peptide Fragments/*pharmacology [MESH] |Protective Agents/pharmacology [MESH] |Random Allocation [MESH] |Rats, Sprague-Dawley [MESH] |Reproducibility of Results [MESH] |Reverse Transcriptase Polymerase Chain Reaction [MESH] |Sleep Apnea, Obstructive/complications [MESH]Angiotensin-(1-7) inhibits inflammation and oxidative stress to reliev(epmc).pdf DeepDyve Pubget Overpricing