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10.15420/AER.2016.4.3 http://scihub22266oqcxt.onion/10.15420/AER.2016.4.3 C5013177!5013177!27617087     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Arrhythm+Electrophysiol+Rev 2016 ; 5 (2): 90-101 Nephropedia Template TP {{tp|p=27617087|t=2016. Arrhythmogenic Cardiomyopathy: Electrical and Structural Phenotypes |pdf=|usr=}}{{27617087}} gab.com Text Arrhythmogenic Cardiomyopathy: Electrical and Structural Phenotypes JO: Arrhythm Electrophysiol Rev http://www.kidney.de/pget.php?&tw=1&pmid=27617087 #FOAvip This overview gives an update on the molecular mechanisms, clinical manifestations, diagnosis and therapy of arrhythmogenic cardiomyopathy (ACM). ACM is mostly hereditary and associated with mutations in genes encoding proteins of the intercalated disc. Three subtypes have been proposed: the classical right-dominant subtype generally referred to as ARVC/D, biventricular forms with early biventricular involvement and left-dominant subtypes with predominant LV involvement. Typical symptoms include palpitations, arrhythmic (pre)syncope and sudden cardiac arrest due to ventricular arrhythmias, which typically occur in athletes. At later stages, heart failure may occur. Diagnosis is established with the 2010 Task Force Criteria (TFC). Modern imaging tools are crucial for ACM diagnosis, including both echocardiography and cardiac magnetic resonance imaging for detecting functional and structural alternations. Of note, structural findings often become visible after electrical alterations, such as premature ventricular beats, ventricular fibrillation (VF) and ventricular tachycardia (VT). 12-lead ECG is important to assess for depolarisation and repolarisation abnormalities, including T-wave inversions as the most common ECG abnormality. Family history and the detection of causative mutations, mostly affecting the desmosome, have been incorporated in the TFC, and stress the importance of cascade family screening. Differential diagnoses include idiopathic right ventricular outflow tract (RVOT) VT, sarcoidosis, congenital heart disease, myocarditis, dilated cardiomyopathy, athlete?s heart, Brugada syndrome and RV infarction. Therapeutic strategies include restriction from endurance and competitive sports, ?-blockers, antiarrhythmic drugs, heart failure medication, implantable cardioverter-defibrillators and endocardial/epicardial catheter ablation. Twit Text FOAVip Arrhythmogenic Cardiomyopathy: Electrical and Structural Phenotypes ????Arrhythm Electrophysiol Rev http://www.kidney.de/pget.php?&tw=1&pmid=27617087 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27617087 #FOAvip English Wikipedia <ref>{{Cite pmid|27617087}}</ref> Arrhythmogenic Cardiomyopathy: Electrical and Structural Phenotypes #MMPMID27617087 Akdis D; Brunckhorst C; Duru F; Saguner AM Arrhythm Electrophysiol Rev 2016[Aug]; 5 (2): 90-101 PMID27617087show ga This overview gives an update on the molecular mechanisms, clinical manifestations, diagnosis and therapy of arrhythmogenic cardiomyopathy (ACM). ACM is mostly hereditary and associated with mutations in genes encoding proteins of the intercalated disc. Three subtypes have been proposed: the classical right-dominant subtype generally referred to as ARVC/D, biventricular forms with early biventricular involvement and left-dominant subtypes with predominant LV involvement. Typical symptoms include palpitations, arrhythmic (pre)syncope and sudden cardiac arrest due to ventricular arrhythmias, which typically occur in athletes. At later stages, heart failure may occur. Diagnosis is established with the 2010 Task Force Criteria (TFC). Modern imaging tools are crucial for ACM diagnosis, including both echocardiography and cardiac magnetic resonance imaging for detecting functional and structural alternations. Of note, structural findings often become visible after electrical alterations, such as premature ventricular beats, ventricular fibrillation (VF) and ventricular tachycardia (VT). 12-lead ECG is important to assess for depolarisation and repolarisation abnormalities, including T-wave inversions as the most common ECG abnormality. Family history and the detection of causative mutations, mostly affecting the desmosome, have been incorporated in the TFC, and stress the importance of cascade family screening. Differential diagnoses include idiopathic right ventricular outflow tract (RVOT) VT, sarcoidosis, congenital heart disease, myocarditis, dilated cardiomyopathy, athlete?s heart, Brugada syndrome and RV infarction. Therapeutic strategies include restriction from endurance and competitive sports, ?-blockers, antiarrhythmic drugs, heart failure medication, implantable cardioverter-defibrillators and endocardial/epicardial catheter ablation. äArrhythmogenic Cardiomyopathy: Electrical and Structural Phenotypes (epmc).pdf DeepDyve Pubget Overpricing