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10.1186/s12882-016-0344-1 http://scihub22266oqcxt.onion/10.1186/s12882-016-0344-1 C5011876!5011876!27596164     free     free     free Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 269.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       BMC+Nephrol 2016 ; 17 (1): ä Nephropedia Template TP {{tp|p=27596164|t=2016. The possible involvement of intestine-derived IgA1: a case of IgA nephropathy associated with Crohn?s disease |pdf=|usr=}}{{27596164}} gab.com Text The possible involvement of intestine-derived IgA1: a case of IgA nephropathy associated with Crohn s disease JO: BMC Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=27596164 #FOAvip Background: A link between IgA nephropathy and Crohn?s disease has recently been reported. Other researchers hypothesize that intestine-derived IgA complexes deposit in glomerular mesangial cells, eliciting IgA nephropathy. Intestinal mucosal plasma cells mainly secrete IgA2. Nevertheless, IgA1 deposition is strongly implicated as being the primary cause of IgA nephropathy. Case presentation: A 46-year-old Japanese man developed IgA nephropathy 29 years ago, following tonsillectomy. As a result, a normal urinalysis was obtained. The patient previously suffered Crohn?s disease followed by urinary occult blood and proteinuria six years ago. Exacerbation of IgA nephropathy was highly suspected. Therefore a renal biopsy was performed. A diagnosis of exacerbation of IgA nephropathy with mesangial cell proliferation and fibrotic cellular crescent was based upon the pathological findings. The patient exhibited a positive clinical course and eventually achieved a remission with immunosuppressive therapy including prednisolone treatment. Immunostaining for the detection of IgA subtypes was performed on both of his kidney and excised ileum. The results revealed IgA1 and IgA2 deposition by submucosal cells in intestine. Furthermore, IgA1 deposition of mesangial areas in the patient?s kidney, indicated an association of IgA1 with the exacerbation of IgA nephropathy. Conclusion: This case represents the possibility that the intestine-derived IgA1 can be the origin of galactose-deficient IgA which is known to cause IgA nephropathy exacerbation. Twit Text FOAVip The possible involvement of intestine-derived IgA1: a case of IgA nephropathy associated with Crohn s disease ????BMC Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=27596164 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27596164 #FOAvip English Wikipedia <ref>{{Cite pmid|27596164}}</ref> The possible involvement of intestine-derived IgA1: a case of IgA nephropathy associated with Crohn?s disease #MMPMID27596164 Terasaka T; Uchida HA; Umebayashi R; Tsukamoto K; Tanaka K; Kitagawa M; Sugiyama H; Tanioka H; Wada J BMC Nephrol 2016[]; 17 (1): ä PMID27596164show ga Background: A link between IgA nephropathy and Crohn?s disease has recently been reported. Other researchers hypothesize that intestine-derived IgA complexes deposit in glomerular mesangial cells, eliciting IgA nephropathy. Intestinal mucosal plasma cells mainly secrete IgA2. Nevertheless, IgA1 deposition is strongly implicated as being the primary cause of IgA nephropathy. Case presentation: A 46-year-old Japanese man developed IgA nephropathy 29 years ago, following tonsillectomy. As a result, a normal urinalysis was obtained. The patient previously suffered Crohn?s disease followed by urinary occult blood and proteinuria six years ago. Exacerbation of IgA nephropathy was highly suspected. Therefore a renal biopsy was performed. A diagnosis of exacerbation of IgA nephropathy with mesangial cell proliferation and fibrotic cellular crescent was based upon the pathological findings. The patient exhibited a positive clinical course and eventually achieved a remission with immunosuppressive therapy including prednisolone treatment. Immunostaining for the detection of IgA subtypes was performed on both of his kidney and excised ileum. The results revealed IgA1 and IgA2 deposition by submucosal cells in intestine. Furthermore, IgA1 deposition of mesangial areas in the patient?s kidney, indicated an association of IgA1 with the exacerbation of IgA nephropathy. Conclusion: This case represents the possibility that the intestine-derived IgA1 can be the origin of galactose-deficient IgA which is known to cause IgA nephropathy exacerbation. äThe possible involvement of intestine-derived IgA1: a case of IgA neph(epmc).pdf DeepDyve Pubget Overpricing