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10.1093/ndt/gfv448 http://scihub22266oqcxt.onion/10.1093/ndt/gfv448 C5009289!5009289 !26908767     free     free     free Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 263.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 263.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26908767 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Nephrol+Dial+Transplant 2016 ; 31 (9 ): 1437-43 Nephropedia Template TP {{tp|p=26908767 |t=2016. Changes in inflammatory biomarkers after renal revascularization in atherosclerotic renal artery stenosis |pdf=|usr=}}{{26908767 }} gab.com Text Changes in inflammatory biomarkers after renal revascularization in atherosclerotic renal artery stenosis JO: Nephrol Dial Transplant http://www.kidney.de/pget.php?&tw=1&pmid=26908767 #FOAvip BACKGROUND: Atherosclerotic renal artery stenosis (ARAS) activates oxidative stress and chronic inflammatory injury. Contrast imaging and endovascular stenting pose potential hazards for acute kidney injury, particularly when superimposed upon reduced kidney perfusion. METHODS: We measured sequential early and long-term changes in circulating inflammatory and injury biomarkers in 12 ARAS subjects subjected to computed tomography imaging and stent revascularization compared with essential hypertensive (EH) subjects of similar age under fixed sodium intake and medication regimens in a clinical research unit. RESULTS: NGAL, TIMP-2, IGFBP7, MCP-1 and TNF-? all were elevated before intervention. Post-stenotic kidney volume, perfusion, blood flow and glomerular filtration rate (GFR) were lower in ARAS than in EH subjects. TIMP-2 and IGFBP7 fell briefly, then rose over 18 h after contrast imaging and stent deployment. Circulating NGAL decreased and remained lower for 27 h. These biomarkers in ARAS returned to baseline after 3 months, while kidney volume, perfusion, blood flow and GFR increased, but remained lower than EH. CONCLUSIONS: These divergent patterns of inflammatory signals are consistent with cell cycle arrest (TIMP-2, IGFBP7) and relative protection from acute kidney injury after imaging and stenting. Sustained basal elevation of circulating and renal venous inflammatory biomarkers support ongoing, possibly episodic, renal stress in ARAS that limits toxicity from stent revascularization. Twit Text FOAVip Changes in inflammatory biomarkers after renal revascularization in atherosclerotic renal artery stenosis ????Nephrol Dial Transplant http://www.kidney.de/pget.php?&tw=1&pmid=26908767 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26908767 #FOAvip English Wikipedia <ref>{{Cite pmid|26908767 }}</ref> Changes in inflammatory biomarkers after renal revascularization in atherosclerotic renal artery stenosis #MMPMID26908767 Wang W ; Saad A ; Herrmann SM ; Eirin Massat A ; McKusick MA ; Misra S ; Lerman LO ; Textor SC Nephrol Dial Transplant 2016[Sep]; 31 (9 ): 1437-43 PMID26908767 show ga BACKGROUND: Atherosclerotic renal artery stenosis (ARAS) activates oxidative stress and chronic inflammatory injury. Contrast imaging and endovascular stenting pose potential hazards for acute kidney injury, particularly when superimposed upon reduced kidney perfusion. METHODS: We measured sequential early and long-term changes in circulating inflammatory and injury biomarkers in 12 ARAS subjects subjected to computed tomography imaging and stent revascularization compared with essential hypertensive (EH) subjects of similar age under fixed sodium intake and medication regimens in a clinical research unit. RESULTS: NGAL, TIMP-2, IGFBP7, MCP-1 and TNF-? all were elevated before intervention. Post-stenotic kidney volume, perfusion, blood flow and glomerular filtration rate (GFR) were lower in ARAS than in EH subjects. TIMP-2 and IGFBP7 fell briefly, then rose over 18 h after contrast imaging and stent deployment. Circulating NGAL decreased and remained lower for 27 h. These biomarkers in ARAS returned to baseline after 3 months, while kidney volume, perfusion, blood flow and GFR increased, but remained lower than EH. CONCLUSIONS: These divergent patterns of inflammatory signals are consistent with cell cycle arrest (TIMP-2, IGFBP7) and relative protection from acute kidney injury after imaging and stenting. Sustained basal elevation of circulating and renal venous inflammatory biomarkers support ongoing, possibly episodic, renal stress in ARAS that limits toxicity from stent revascularization. |Aged [MESH] |Atherosclerosis/*complications [MESH] |Biomarkers/*blood [MESH] |Chemokine CCL2/blood [MESH] |Female [MESH] |Glomerular Filtration Rate [MESH] |Humans [MESH] |Inflammation/blood/*diagnosis/etiology [MESH] |Male [MESH] |Middle Aged [MESH] |Oxidative Stress [MESH] |Prospective Studies [MESH] |Renal Artery Obstruction/diagnosis/etiology/*therapy [MESH] |Stents [MESH] |Tissue Inhibitor of Metalloproteinase-2/blood [MESH]Changes in inflammatory biomarkers after renal revascularization in (epmc).pdf DeepDyve Pubget Overpricing