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10.3390/ijms17081312 http://scihub22266oqcxt.onion/10.3390/ijms17081312 C5000709!5000709!27529213     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Int+J+Mol+Sci 2016 ; 17 (8): ä Nephropedia Template TP {{tp|p=27529213|t=2016. The Potential Role of Kallistatin in the Development of Abdominal Aortic Aneurysm |pdf=|usr=}}{{27529213}} gab.com Text The Potential Role of Kallistatin in the Development of Abdominal Aortic Aneurysm JO: Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=27529213 #FOAvip Abdominal aortic aneurysm (AAA) is a vascular condition that causes permanent dilation of the abdominal aorta, which can lead to death due to aortic rupture. The only treatment for AAA is surgical repair, and there is no current drug treatment for AAA. Aortic inflammation, vascular smooth muscle cell apoptosis, angiogenesis, oxidative stress and vascular remodeling are implicated in AAA pathogenesis. Kallistatin is a serine proteinase inhibitor, which has been shown to have a variety of functions, potentially relevant in AAA pathogenesis. Kallistatin has been reported to have inhibitory effects on tumor necrosis factor alpha (TNF-?) signaling induced oxidative stress and apoptosis. Kallistatin also inhibits vascular endothelial growth factor (VEGF) and Wnt canonical signaling, which promote inflammation, angiogenesis, and vascular remodeling in various pre-clinical experimental models. This review explores the potential protective role of kallistatin in AAA pathogenesis. Twit Text FOAVip The Potential Role of Kallistatin in the Development of Abdominal Aortic Aneurysm ????Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=27529213 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27529213 #FOAvip English Wikipedia <ref>{{Cite pmid|27529213}}</ref> The Potential Role of Kallistatin in the Development of Abdominal Aortic Aneurysm #MMPMID27529213 Li J; Krishna SM; Golledge J Int J Mol Sci 2016[Aug]; 17 (8): ä PMID27529213show ga Abdominal aortic aneurysm (AAA) is a vascular condition that causes permanent dilation of the abdominal aorta, which can lead to death due to aortic rupture. The only treatment for AAA is surgical repair, and there is no current drug treatment for AAA. Aortic inflammation, vascular smooth muscle cell apoptosis, angiogenesis, oxidative stress and vascular remodeling are implicated in AAA pathogenesis. Kallistatin is a serine proteinase inhibitor, which has been shown to have a variety of functions, potentially relevant in AAA pathogenesis. Kallistatin has been reported to have inhibitory effects on tumor necrosis factor alpha (TNF-?) signaling induced oxidative stress and apoptosis. Kallistatin also inhibits vascular endothelial growth factor (VEGF) and Wnt canonical signaling, which promote inflammation, angiogenesis, and vascular remodeling in various pre-clinical experimental models. This review explores the potential protective role of kallistatin in AAA pathogenesis. äThe Potential Role of Kallistatin in the Development of Abdominal Aort(epmc).pdf DeepDyve Pubget Overpricing