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10.3389/fphar.2016.00264 http://scihub22266oqcxt.onion/10.3389/fphar.2016.00264 C4992728!4992728!27597829     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Front+Pharmacol 2016 ; 7 (ä): ä Nephropedia Template TP {{tp|p=27597829|t=2016. CB2 and GPR55 Receptors as Therapeutic Targets for Systemic Immune Dysregulation |pdf=|usr=}}{{27597829}} gab.com Text CB2 and GPR55 Receptors as Therapeutic Targets for Systemic Immune Dysregulation JO: Front Pharmacol http://www.kidney.de/pget.php?&tw=1&pmid=27597829 #FOAvip The endocannabinoid system (ECS) is involved in many physiological processes and has been suggested to play a critical role in the immune response and the central nervous system (CNS). Therefore, ECS modulation has potential therapeutic effects on immune dysfunctional disorders, such as sepsis and CNS injury-induced immunodeficiency syndrome (CIDS). In sepsis, excessive release of pro- and anti-inflammatory mediators results in multi-organ dysfunction, failure, and death. In CIDS, an acute CNS injury dysregulates a normally well-balanced interplay between CNS and the immune system, leading to increased patients? susceptibility to infections. In this review, we will discuss potential therapeutic modulation of the immune response in sepsis and CNS injury by manipulation of the ECS representing a novel target for immunotherapy. Twit Text FOAVip CB2 and GPR55 Receptors as Therapeutic Targets for Systemic Immune Dysregulation ????Front Pharmacol http://www.kidney.de/pget.php?&tw=1&pmid=27597829 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27597829 #FOAvip English Wikipedia <ref>{{Cite pmid|27597829}}</ref> CB2 and GPR55 Receptors as Therapeutic Targets for Systemic Immune Dysregulation #MMPMID27597829 Zhou J; Burkovskiy I; Yang H; Sardinha J; Lehmann C Front Pharmacol 2016[]; 7 (ä): ä PMID27597829show ga The endocannabinoid system (ECS) is involved in many physiological processes and has been suggested to play a critical role in the immune response and the central nervous system (CNS). Therefore, ECS modulation has potential therapeutic effects on immune dysfunctional disorders, such as sepsis and CNS injury-induced immunodeficiency syndrome (CIDS). In sepsis, excessive release of pro- and anti-inflammatory mediators results in multi-organ dysfunction, failure, and death. In CIDS, an acute CNS injury dysregulates a normally well-balanced interplay between CNS and the immune system, leading to increased patients? susceptibility to infections. In this review, we will discuss potential therapeutic modulation of the immune response in sepsis and CNS injury by manipulation of the ECS representing a novel target for immunotherapy. äCB2 and GPR55 Receptors as Therapeutic Targets for Systemic Immune Dys(epmc).pdf DeepDyve Pubget Overpricing