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10.1158/1078-0432.CCR-16-0038 http://scihub22266oqcxt.onion/10.1158/1078-0432.CCR-16-0038 C4946572!4946572!26957555     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Clin+Cancer+Res 2016 ; 22 (9): 2099-101 Nephropedia Template TP {{tp|p=26957555|t=2016. Pharmacology in the Era of Targeted Therapies: The Case of PI3K Inhibitors |pdf=|usr=}}{{26957555}} gab.com Text Pharmacology in the Era of Targeted Therapies: The Case of PI3K Inhibitors JO: Clin Cancer Res http://www.kidney.de/pget.php?&tw=1&pmid=26957555 #FOAvip The Phosphoinositide-3-kinase (PI3K) pathway is often aberrantly activated in Estrogen Receptor positive (ER+) breast cancer and therapies combining PI3K inhibitors and anti-estrogens are under clinical development. Given that many PI3K inhibitors have substantial toxicities with continuous dosing and that alternate dosing schedules are equally active, further clinical exploration is warranted. Twit Text FOAVip Pharmacology in the Era of Targeted Therapies: The Case of PI3K Inhibitors ????Clin Cancer Res http://www.kidney.de/pget.php?&tw=1&pmid=26957555 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26957555 #FOAvip English Wikipedia <ref>{{Cite pmid|26957555}}</ref> Pharmacology in the Era of Targeted Therapies: The Case of PI3K Inhibitors #MMPMID26957555 Toska E; Baselga J Clin Cancer Res 2016[May]; 22 (9): 2099-101 PMID26957555show ga The Phosphoinositide-3-kinase (PI3K) pathway is often aberrantly activated in Estrogen Receptor positive (ER+) breast cancer and therapies combining PI3K inhibitors and anti-estrogens are under clinical development. Given that many PI3K inhibitors have substantial toxicities with continuous dosing and that alternate dosing schedules are equally active, further clinical exploration is warranted. äPharmacology in the Era of Targeted Therapies: The Case of PI3K Inhibi(epmc).pdf DeepDyve Pubget Overpricing