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10.1111/nyas.13091 http://scihub22266oqcxt.onion/10.1111/nyas.13091 C4940235!4940235!27303906     free     free     free       Ann+N+Y+Acad+Sci 2016 ; 1374 (1): 159-67 Nephropedia Template TP {{tp|p=27303906|t=2016. Chlorine-induced cardiopulmonary injury |pdf=|usr=}}{{27303906}} gab.com Text Chlorine-induced cardiopulmonary injury JO: Ann N Y Acad Sci http://www.kidney.de/pget.php?&tw=1&pmid=27303906 #FOAvip Chlorine (Cl2) is utilized worldwide for a diverse range of industrial applications, including pulp bleaching, sanitation, and pharmaceutical development. Though Cl2 has widespread use, little is known regarding the mechanisms of toxicity associated with Cl2 exposure, which occurs during industrial accidents or acts of terrorism. Previous instances of Cl2 exposure have led to reported episodes of respiratory distress that result in high morbidity and mortality. Furthermore, studies suggest that acute Cl2 exposure also results in systemic vascular injury and subsequent myocardial contractile dysfunction. Here we review both lung and cardiac pathology associated with acute Cl2 inhalation and discuss recently published data that suggests that mitochondrial dysfunction underlies the pathogenesis of Cl2-induced toxicity. Lastly, we discuss our findings that suggest that upregulation of autophagy protects against Cl2-induced lung inflammation and can be a potential therapeutic target for ameliorating the toxic effects of Cl2 exposure. Twit Text FOAVip Chlorine-induced cardiopulmonary injury ????Ann N Y Acad Sci http://www.kidney.de/pget.php?&tw=1&pmid=27303906 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27303906 #FOAvip English Wikipedia <ref>{{Cite pmid|27303906}}</ref> Chlorine-induced cardiopulmonary injury #MMPMID27303906 Carlisle M; Lam A; Svendsen ER; Aggarwal S; Matalon S Ann N Y Acad Sci 2016[Jun]; 1374 (1): 159-67 PMID27303906show ga Chlorine (Cl2) is utilized worldwide for a diverse range of industrial applications, including pulp bleaching, sanitation, and pharmaceutical development. Though Cl2 has widespread use, little is known regarding the mechanisms of toxicity associated with Cl2 exposure, which occurs during industrial accidents or acts of terrorism. Previous instances of Cl2 exposure have led to reported episodes of respiratory distress that result in high morbidity and mortality. Furthermore, studies suggest that acute Cl2 exposure also results in systemic vascular injury and subsequent myocardial contractile dysfunction. Here we review both lung and cardiac pathology associated with acute Cl2 inhalation and discuss recently published data that suggests that mitochondrial dysfunction underlies the pathogenesis of Cl2-induced toxicity. Lastly, we discuss our findings that suggest that upregulation of autophagy protects against Cl2-induced lung inflammation and can be a potential therapeutic target for ameliorating the toxic effects of Cl2 exposure. äChlorine-induced cardiopulmonary injury (epmc).pdf DeepDyve Pubget Overpricing