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10.1038/srep28573 http://scihub22266oqcxt.onion/10.1038/srep28573 C4926115!4926115 !27353032     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27353032 &cmd=llinks): Failed to open stream: HTTP request failed! 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Enlarged colitogenic T cell population paradoxically supports colitis prevention through the B-lymphocyte-dependent peripheral generation of CD4(+)Foxp3(+) Treg cells |pdf=|usr=}}{{27353032 }} gab.com Text Enlarged colitogenic T cell population paradoxically supports colitis prevention through the B-lymphocyte-dependent peripheral generation of CD4(+)Foxp3(+) Treg cells JO: Sci Rep http://www.kidney.de/pget.php?&tw=1&pmid=27353032 #FOAvip Intestinal inflammation can be induced by the reconstitution of T/B cell-deficient mice with low numbers of CD4(+) T lymphocytes depleted of CD25(+)Foxp3(+) regulatory T cells (Treg). Using RAG-knockout mice as recipients of either splenocytes exclusively depleted of CD25(+) cells or FACS-purified CD4(+)CD25(-)Foxp3(-) T cells, we found that the augmentation of potentially colitogenic naïve T cell numbers in the inoculum was unexpectedly beneficial for the suppression of colon disease and maintenance of immune homeostasis. Protection against T cell-mediated colitis correlated with a significant increment in the frequency of peripherally-induced CD4(+)CD25(+)Foxp3(+) T (pTreg) cells, especially in the mesenteric lymph nodes, an effect that required the presence of B cells and CD4(+)CD25(-)Foxp3(+) cells in physiological proportions. Our findings support a model whereby the interplay between B lymphocytes and a diversified naïve T cell repertoire is critical for the generation of CD4(+)CD25(+)Foxp3(+) pTreg cells and colitis suppression. Twit Text FOAVip Enlarged colitogenic T cell population paradoxically supports colitis prevention through the B-lymphocyte-dependent peripheral generation of CD4(+)Foxp3(+) Treg cells ????Sci Rep http://www.kidney.de/pget.php?&tw=1&pmid=27353032 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27353032 #FOAvip English Wikipedia <ref>{{Cite pmid|27353032 }}</ref> Enlarged colitogenic T cell population paradoxically supports colitis prevention through the B-lymphocyte-dependent peripheral generation of CD4(+)Foxp3(+) Treg cells #MMPMID27353032 do Canto FB ; Campos SM ; Granato A ; da Silva RF ; de Paiva LS ; Nóbrega A ; Bellio M ; Fucs R Sci Rep 2016[Jun]; 6 (ä): 28573 PMID27353032 show ga Intestinal inflammation can be induced by the reconstitution of T/B cell-deficient mice with low numbers of CD4(+) T lymphocytes depleted of CD25(+)Foxp3(+) regulatory T cells (Treg). Using RAG-knockout mice as recipients of either splenocytes exclusively depleted of CD25(+) cells or FACS-purified CD4(+)CD25(-)Foxp3(-) T cells, we found that the augmentation of potentially colitogenic naïve T cell numbers in the inoculum was unexpectedly beneficial for the suppression of colon disease and maintenance of immune homeostasis. Protection against T cell-mediated colitis correlated with a significant increment in the frequency of peripherally-induced CD4(+)CD25(+)Foxp3(+) T (pTreg) cells, especially in the mesenteric lymph nodes, an effect that required the presence of B cells and CD4(+)CD25(-)Foxp3(+) cells in physiological proportions. Our findings support a model whereby the interplay between B lymphocytes and a diversified naïve T cell repertoire is critical for the generation of CD4(+)CD25(+)Foxp3(+) pTreg cells and colitis suppression. |*Models, Immunological [MESH] |Animals [MESH] |B-Lymphocytes/*immunology/pathology [MESH] |Colitis/genetics/*immunology/pathology/*prevention & control [MESH] |Lymph Nodes/immunology/pathology [MESH] |Mesentery/immunology/pathology [MESH] |Mice [MESH] |Mice, Knockout [MESH]Enlarged colitogenic T cell population paradoxically supports colitis (epmc).pdf DeepDyve Pubget Overpricing