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10.1016/j.tem.2016.04.006 http://scihub22266oqcxt.onion/10.1016/j.tem.2016.04.006 C4912878!4912878!27160798     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Trends+Endocrinol+Metab 2016 ; 27 (7): 484-92 Nephropedia Template TP {{tp|p=27160798|t=2016. Map4k4 signaling nodes in metabolic and cardiovascular diseases |pdf=|usr=}}{{27160798}} gab.com Text Map4k4 signaling nodes in metabolic and cardiovascular diseases JO: Trends Endocrinol Metab http://www.kidney.de/pget.php?&tw=1&pmid=27160798 #FOAvip Map4k4, originally identified in siRNA screens and characterized by tissue specific gene deletions, is emerging as a regulator of glucose homeostasis and cardiovascular health. Recent studies have shown that Map4k4 gene ablation or inhibition of its kinase activity attenuates hyperglycemia and plaque formation in mouse models of insulin resistance and atherosclerosis, and suggest roles for Map4k4 in multiple signaling systems, including NF?B activation, small GTPase regulation, the Hippo cascade, and regulation of cell dynamics by FERM domain proteins. This new and promising area of inquiry raises key questions that need to be addressed in the future, such as defining which of the above or other effectors mediate Map4k4 control of metabolic and vascular functions, and identifying upstream activators of Map4k4. Twit Text FOAVip Map4k4 signaling nodes in metabolic and cardiovascular diseases ????Trends Endocrinol Metab http://www.kidney.de/pget.php?&tw=1&pmid=27160798 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27160798 #FOAvip English Wikipedia <ref>{{Cite pmid|27160798}}</ref> Map4k4 signaling nodes in metabolic and cardiovascular diseases #MMPMID27160798 Virbasius JV; Czech MP Trends Endocrinol Metab 2016[Jul]; 27 (7): 484-92 PMID27160798show ga Map4k4, originally identified in siRNA screens and characterized by tissue specific gene deletions, is emerging as a regulator of glucose homeostasis and cardiovascular health. Recent studies have shown that Map4k4 gene ablation or inhibition of its kinase activity attenuates hyperglycemia and plaque formation in mouse models of insulin resistance and atherosclerosis, and suggest roles for Map4k4 in multiple signaling systems, including NF?B activation, small GTPase regulation, the Hippo cascade, and regulation of cell dynamics by FERM domain proteins. This new and promising area of inquiry raises key questions that need to be addressed in the future, such as defining which of the above or other effectors mediate Map4k4 control of metabolic and vascular functions, and identifying upstream activators of Map4k4. äMap4k4 signaling nodes in metabolic and cardiovascular diseases (epmc).pdf DeepDyve Pubget Overpricing