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10.1155/2016/6489012 http://scihub22266oqcxt.onion/10.1155/2016/6489012 C4904645!4904645!27446858     free     free     free Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Can+J+Gastroenterol+Hepatol 2016 ; 2016 (ä): ä Nephropedia Template TP {{tp|p=27446858|t=2016. Role of NLRP3 Inflammasome in the Progression of NAFLD to NASH |pdf=|usr=}}{{27446858}} gab.com Text Role of NLRP3 Inflammasome in the Progression of NAFLD to NASH JO: Can J Gastroenterol Hepatol http://www.kidney.de/pget.php?&tw=1&pmid=27446858 #FOAvip Nonalcoholic fatty liver disease (NAFLD) has been recognized as a major public health problem worldwide. Nonalcoholic steatohepatitis (NASH) is an advanced form of NAFLD that may progress to cirrhosis and hepatocellular carcinoma. The pathogenesis of disease progression from NAFLD to NASH has not been fully understood. Immunological mechanisms that have been increasingly recognized in the disease progression include defects in innate immunity, adaptive immunity, Toll-like receptor (TLR) signaling, and gut-liver axis. The NLRP3 inflammasome is an intracellular multiprotein complex involved in the production of mature interleukin 1-beta (IL-1?) and induces metabolic inflammation. NLRP3 inflammasome has been recently demonstrated to play a crucial role in the progression of NASH. This review highlights the recent findings linking NLRP3 inflammasome to the progression of NASH. Twit Text FOAVip Role of NLRP3 Inflammasome in the Progression of NAFLD to NASH ????Can J Gastroenterol Hepatol http://www.kidney.de/pget.php?&tw=1&pmid=27446858 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27446858 #FOAvip English Wikipedia <ref>{{Cite pmid|27446858}}</ref> Role of NLRP3 Inflammasome in the Progression of NAFLD to NASH #MMPMID27446858 Wan X; Xu C; Yu C; Li Y Can J Gastroenterol Hepatol 2016[]; 2016 (ä): ä PMID27446858show ga Nonalcoholic fatty liver disease (NAFLD) has been recognized as a major public health problem worldwide. Nonalcoholic steatohepatitis (NASH) is an advanced form of NAFLD that may progress to cirrhosis and hepatocellular carcinoma. The pathogenesis of disease progression from NAFLD to NASH has not been fully understood. Immunological mechanisms that have been increasingly recognized in the disease progression include defects in innate immunity, adaptive immunity, Toll-like receptor (TLR) signaling, and gut-liver axis. The NLRP3 inflammasome is an intracellular multiprotein complex involved in the production of mature interleukin 1-beta (IL-1?) and induces metabolic inflammation. NLRP3 inflammasome has been recently demonstrated to play a crucial role in the progression of NASH. This review highlights the recent findings linking NLRP3 inflammasome to the progression of NASH. äRole of NLRP3 Inflammasome in the Progression of NAFLD to NASH (epmc).pdf DeepDyve Pubget Overpricing