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10.1097/YCO.0000000000000244 http://scihub22266oqcxt.onion/10.1097/YCO.0000000000000244 C4901383!4901383 !26900672     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26900672 &cmd=llinks): Failed to open stream: HTTP request failed! 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Gamma band oscillations: a key to understanding schizophrenia symptoms and neural circuit abnormalities |pdf=|usr=}}{{26900672 }} gab.com Text Gamma band oscillations: a key to understanding schizophrenia symptoms and neural circuit abnormalities JO: Curr Opin Psychiatry http://www.kidney.de/pget.php?&tw=1&pmid=26900672 #FOAvip PURPOSE OF REVIEW: We review our current understanding of abnormal ? band oscillations in schizophrenia, their association with symptoms and the underlying cortical circuit abnormality, with a particular focus on the role of fast-spiking parvalbumin gamma-aminobutyric acid (GABA) neurons in the disease state. RECENT FINDINGS: Clinical electrophysiological studies of schizophrenia patients and pharmacological models of the disorder show an increase in spontaneous ? band activity (not stimulus-evoked) measures. These findings provide a crucial link between preclinical and clinical work examining the role of ? band activity in schizophrenia. MRI-based experiments measuring cortical GABA provides evidence supporting impaired GABAergic neurotransmission in schizophrenia patients, which is correlated with ? band activity level. Several studies suggest that stimulation of the cortical circuitry, directly or via subcortical structures, has the potential to modulate cortical ? activity, and improve cognitive function. SUMMARY: Abnormal ? band activity is observed in patients with schizophrenia and disease models in animals, and is suggested to underlie the psychosis and cognitive/perceptual deficits. Convergent evidence from both clinical and preclinical studies suggest the central factor in ? band abnormalities is impaired GABAergic neurotransmission, particularly in a subclass of neurons which express parvalbumin. Rescue of ? band abnormalities presents an intriguing option for therapeutic intervention. Twit Text FOAVip Gamma band oscillations: a key to understanding schizophrenia symptoms and neural circuit abnormalities ????Curr Opin Psychiatry http://www.kidney.de/pget.php?&tw=1&pmid=26900672 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26900672 #FOAvip English Wikipedia <ref>{{Cite pmid|26900672 }}</ref> Gamma band oscillations: a key to understanding schizophrenia symptoms and neural circuit abnormalities #MMPMID26900672 McNally JM ; McCarley RW Curr Opin Psychiatry 2016[May]; 29 (3 ): 202-10 PMID26900672 show ga PURPOSE OF REVIEW: We review our current understanding of abnormal ? band oscillations in schizophrenia, their association with symptoms and the underlying cortical circuit abnormality, with a particular focus on the role of fast-spiking parvalbumin gamma-aminobutyric acid (GABA) neurons in the disease state. RECENT FINDINGS: Clinical electrophysiological studies of schizophrenia patients and pharmacological models of the disorder show an increase in spontaneous ? band activity (not stimulus-evoked) measures. These findings provide a crucial link between preclinical and clinical work examining the role of ? band activity in schizophrenia. MRI-based experiments measuring cortical GABA provides evidence supporting impaired GABAergic neurotransmission in schizophrenia patients, which is correlated with ? band activity level. Several studies suggest that stimulation of the cortical circuitry, directly or via subcortical structures, has the potential to modulate cortical ? activity, and improve cognitive function. SUMMARY: Abnormal ? band activity is observed in patients with schizophrenia and disease models in animals, and is suggested to underlie the psychosis and cognitive/perceptual deficits. Convergent evidence from both clinical and preclinical studies suggest the central factor in ? band abnormalities is impaired GABAergic neurotransmission, particularly in a subclass of neurons which express parvalbumin. Rescue of ? band abnormalities presents an intriguing option for therapeutic intervention. |*Synaptic Transmission [MESH] |Animals [MESH] |Cognition Disorders/metabolism/physiopathology [MESH] |Humans [MESH] |Neurons/*metabolism [MESH] |Parvalbumins/*metabolism [MESH] |Psychotic Disorders/metabolism/physiopathology [MESH] |Schizophrenia/*metabolism/*physiopathology [MESH] |Schizophrenic Psychology [MESH]Gamma band oscillations: a key to understanding schizophrenia symptoms(epmc).pdf DeepDyve Pubget Overpricing