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10.1074/jbc.M116.724690 http://scihub22266oqcxt.onion/10.1074/jbc.M116.724690 C4900260!4900260 !27033704     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27033704 &cmd=llinks): Failed to open stream: HTTP request failed! 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Oxidative Stress and Nuclear Factor ?B (NF-?B) Increase Peritoneal Filtration and Contribute to Ascites Formation in Nephrotic Syndrome |pdf=|usr=}}{{27033704 }} gab.com Text Oxidative Stress and Nuclear Factor B (NF- B) Increase Peritoneal Filtration and Contribute to Ascites Formation in Nephrotic Syndrome JO: J Biol Chem http://www.kidney.de/pget.php?&tw=1&pmid=27033704 #FOAvip Water accumulation in the interstitium (edema) and the peritoneum (ascites) of nephrotic patients is classically thought to stem from the prevailing low plasma albumin concentration and the decreased transcapillary oncotic pressure gradient. However, several clinical and experimental observations suggest that it might also stem from changes in capillary permeability. We addressed this hypothesis by studying the peritoneum permeability of rats with puromycin aminonucleoside-induced nephrotic syndrome. The peritoneum of puromycin aminonucleoside rats displayed an increase in the water filtration coefficient of paracellular and transcellular pathways, and a decrease in the reflection coefficient to proteins. It also displayed oxidative stress and subsequent activation of NF-?B. Scavenging of reactive oxygen species and inhibition of NF-?B prevented the changes in the water permeability and reflection coefficient to proteins and reduced the volume of ascites by over 50%. Changes in water permeability were associated with the overexpression of the water channel aquaporin 1, which was prevented by reactive oxygen species scavenging and inhibition of NF-?B. In conclusion, nephrotic syndrome is associated with an increased filtration coefficient of the peritoneum and a decreased reflection coefficient to proteins. These changes, which account for over half of ascite volume, are triggered by oxidative stress and subsequent activation of NF-?B. Twit Text FOAVip Oxidative Stress and Nuclear Factor B (NF- B) Increase Peritoneal Filtration and Contribute to Ascites Formation in Nephrotic Syndrome ????J Biol Chem http://www.kidney.de/pget.php?&tw=1&pmid=27033704 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27033704 #FOAvip English Wikipedia <ref>{{Cite pmid|27033704 }}</ref> Oxidative Stress and Nuclear Factor ?B (NF-?B) Increase Peritoneal Filtration and Contribute to Ascites Formation in Nephrotic Syndrome #MMPMID27033704 Udwan K ; Brideau G ; Fila M ; Edwards A ; Vogt B ; Doucet A J Biol Chem 2016[May]; 291 (21 ): 11105-13 PMID27033704 show ga Water accumulation in the interstitium (edema) and the peritoneum (ascites) of nephrotic patients is classically thought to stem from the prevailing low plasma albumin concentration and the decreased transcapillary oncotic pressure gradient. However, several clinical and experimental observations suggest that it might also stem from changes in capillary permeability. We addressed this hypothesis by studying the peritoneum permeability of rats with puromycin aminonucleoside-induced nephrotic syndrome. The peritoneum of puromycin aminonucleoside rats displayed an increase in the water filtration coefficient of paracellular and transcellular pathways, and a decrease in the reflection coefficient to proteins. It also displayed oxidative stress and subsequent activation of NF-?B. Scavenging of reactive oxygen species and inhibition of NF-?B prevented the changes in the water permeability and reflection coefficient to proteins and reduced the volume of ascites by over 50%. Changes in water permeability were associated with the overexpression of the water channel aquaporin 1, which was prevented by reactive oxygen species scavenging and inhibition of NF-?B. In conclusion, nephrotic syndrome is associated with an increased filtration coefficient of the peritoneum and a decreased reflection coefficient to proteins. These changes, which account for over half of ascite volume, are triggered by oxidative stress and subsequent activation of NF-?B. |*Ascites/chemically induced/metabolism/pathology [MESH] |*Nephrotic Syndrome/chemically induced/metabolism/pathology [MESH] |*Peritoneum/metabolism/pathology [MESH] |Animals [MESH] |Aquaporin 1/metabolism [MESH] |Humans [MESH] |Male [MESH] |NF-kappa B/*metabolism [MESH] |Oxidative Stress/*drug effects [MESH] |Puromycin Aminonucleoside/*adverse effects/pharmacology [MESH] |Rats [MESH]Oxidative Stress and Nuclear Factor ?B (NF-?B) Increase Peritoneal Fil(epmc).pdf DeepDyve Pubget Overpricing