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10.1016/j.ccep.2016.01.002 http://scihub22266oqcxt.onion/10.1016/j.ccep.2016.01.002 C4894371!4894371!27261821     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27261821&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Card+Electrophysiol+Clin 2016 ; 8 (2): 275-84 Nephropedia Template TP {{tp|p=27261821|t=2016. Molecular Basis of Cardiac Delayed Rectifier K+ Channel Function and Pharmacology |pdf=|usr=}}{{27261821}} gab.com Text Molecular Basis of Cardiac Delayed Rectifier K+ Channel Function and Pharmacology JO: Card Electrophysiol Clin http://www.kidney.de/pget.php?&tw=1&pmid=27261821 #FOAvip Human cardiomyocytes express three distinct types of delayed rectifier potassium channels. hERG1 channels conduct the rapidly activating current IKr, KCNQ1/KCNE1 channels conduct the slowly activating current IKs, and Kv1.5 channels conduct an ultrarapid activating current IKur. Here we provide a general overview of the mechanistic and structural basis of ion selectivity, gating and pharmacology of the three types of cardiac delayed rectifier K+ channels. Most blockers bind to S6 residues that line the central cavity of the channel, whereas activators interact with the channel at four symmetrical binding sites outside the cavity. Twit Text FOAVip Molecular Basis of Cardiac Delayed Rectifier K+ Channel Function and Pharmacology ????Card Electrophysiol Clin http://www.kidney.de/pget.php?&tw=1&pmid=27261821 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27261821 #FOAvip English Wikipedia <ref>{{Cite pmid|27261821}}</ref> Molecular Basis of Cardiac Delayed Rectifier K+ Channel Function and Pharmacology #MMPMID27261821 Wu W; Sanguinetti MC Card Electrophysiol Clin 2016[Jun]; 8 (2): 275-84 PMID27261821show ga Human cardiomyocytes express three distinct types of delayed rectifier potassium channels. hERG1 channels conduct the rapidly activating current IKr, KCNQ1/KCNE1 channels conduct the slowly activating current IKs, and Kv1.5 channels conduct an ultrarapid activating current IKur. Here we provide a general overview of the mechanistic and structural basis of ion selectivity, gating and pharmacology of the three types of cardiac delayed rectifier K+ channels. Most blockers bind to S6 residues that line the central cavity of the channel, whereas activators interact with the channel at four symmetrical binding sites outside the cavity. äMolecular Basis of Cardiac Delayed Rectifier K+ Channel Function and P(epmc).pdf DeepDyve Pubget Overpricing