Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1016/j.ecl.2016.01.006 http://scihub22266oqcxt.onion/10.1016/j.ecl.2016.01.006 C4889872!4889872!27241967     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Endocrinol+Metab+Clin+North+Am 2016 ; 45 (2): 311-28 Nephropedia Template TP {{tp|p=27241967|t=2016. Diagnosis and clinical genetics of Cushing syndrome in pediatrics |pdf=|usr=}}{{27241967}} gab.com Text Diagnosis and clinical genetics of Cushing syndrome in pediatrics JO: Endocrinol Metab Clin North Am http://www.kidney.de/pget.php?&tw=1&pmid=27241967 #FOAvip Endogenous Cushing syndrome (CS) in pediatrics is rare; it may be caused by tumors that produce corticotropin (ACTH) in the pituitary gland (this form of CS is called Cushing disease) or elsewhere (ectopic CS), tumors that produce corticotropin-releasing hormone (CRH) anywhere (mostly neuroendocrine tissues), and finally adrenocortical masses that produce cortisol, such as adrenocortical cancer (ACC) or adenomas, and bilateral adrenocortical hypeprlasia (BAHs). ACC is a very rare cause of CS in children but should be excluded first, especially among younger patients. CS in children is often caused by germline or somatic mutations in an expanding list of genes with implications for the prognosis of the patients and for their families. CS should be early recognized in children; otherwise, it can lead to significant morbidity and mortality. All patients with suspected CS should be referred to specialized clinical centers for work-up; these centers should have access to experienced endocrine and neurological surgeons. Twit Text FOAVip Diagnosis and clinical genetics of Cushing syndrome in pediatrics ????Endocrinol Metab Clin North Am http://www.kidney.de/pget.php?&tw=1&pmid=27241967 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27241967 #FOAvip English Wikipedia <ref>{{Cite pmid|27241967}}</ref> Diagnosis and clinical genetics of Cushing syndrome in pediatrics #MMPMID27241967 Stratakis CA Endocrinol Metab Clin North Am 2016[Jun]; 45 (2): 311-28 PMID27241967show ga Endogenous Cushing syndrome (CS) in pediatrics is rare; it may be caused by tumors that produce corticotropin (ACTH) in the pituitary gland (this form of CS is called Cushing disease) or elsewhere (ectopic CS), tumors that produce corticotropin-releasing hormone (CRH) anywhere (mostly neuroendocrine tissues), and finally adrenocortical masses that produce cortisol, such as adrenocortical cancer (ACC) or adenomas, and bilateral adrenocortical hypeprlasia (BAHs). ACC is a very rare cause of CS in children but should be excluded first, especially among younger patients. CS in children is often caused by germline or somatic mutations in an expanding list of genes with implications for the prognosis of the patients and for their families. CS should be early recognized in children; otherwise, it can lead to significant morbidity and mortality. All patients with suspected CS should be referred to specialized clinical centers for work-up; these centers should have access to experienced endocrine and neurological surgeons. äDiagnosis and clinical genetics of Cushing syndrome in pediatrics (epmc).pdf DeepDyve Pubget Overpricing