Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1186/s40348-016-0049-3 http://scihub22266oqcxt.onion/10.1186/s40348-016-0049-3 C4889530!4889530!27251607     free     free     free       Mol+Cell+Pediatr 2016 ; 3 (ä): ä Nephropedia Template TP {{tp|p=27251607|t=2016. Mechanism of human rhinovirus infections |pdf=|usr=}}{{27251607}} gab.com Text Mechanism of human rhinovirus infections JO: Mol Cell Pediatr http://www.kidney.de/pget.php?&tw=1&pmid=27251607 #FOAvip About 150 human rhinovirus serotypes are responsible for more than 50 % of recurrent upper respiratory infections. Despite having similar 3D structures, some bind members of the low-density lipoprotein receptor family, some ICAM-1, and some use CDHR3 for host cell infection. This is also reflected in the pathways exploited for cellular entry. We found that even rhinovirus serotypes binding the same receptor can travel along different endocytic pathways and release their RNA genome into the cytosol at different locations. How this may account for distinct immune responses elicited by various rhinoviruses and the observed symptoms of the common cold is briefly discussed. Twit Text FOAVip Mechanism of human rhinovirus infections ????Mol Cell Pediatr http://www.kidney.de/pget.php?&tw=1&pmid=27251607 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27251607 #FOAvip English Wikipedia <ref>{{Cite pmid|27251607}}</ref> Mechanism of human rhinovirus infections #MMPMID27251607 Blaas D; Fuchs R Mol Cell Pediatr 2016[Dec]; 3 (ä): ä PMID27251607show ga About 150 human rhinovirus serotypes are responsible for more than 50 % of recurrent upper respiratory infections. Despite having similar 3D structures, some bind members of the low-density lipoprotein receptor family, some ICAM-1, and some use CDHR3 for host cell infection. This is also reflected in the pathways exploited for cellular entry. We found that even rhinovirus serotypes binding the same receptor can travel along different endocytic pathways and release their RNA genome into the cytosol at different locations. How this may account for distinct immune responses elicited by various rhinoviruses and the observed symptoms of the common cold is briefly discussed. äMechanism of human rhinovirus infections (epmc).pdf DeepDyve Pubget Overpricing