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10.1155/2016/7351964

http://scihub22266oqcxt.onion/10.1155/2016/7351964
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C4886048!4886048!27294131
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suck abstract from ncbi


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pmid27294131      Biomed+Res+Int 2016 ; 2016 (ä): ä
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  • Infection-Related Focal Segmental Glomerulosclerosis in Children #MMPMID27294131
  • Dettmar AK; Oh J
  • Biomed Res Int 2016[]; 2016 (ä): ä PMID27294131show ga
  • Focal segmental glomerulosclerosis (FSGS) is the most common cause of steroid resistant nephrotic syndrome in children. It describes a unique histological picture of glomerular damage resulting from several causes. In the majority of patients the causing agent is still unknown, but in some cases viral association is evident. In adults, the most established FSGS causing virus is the human immune-deficiency virus, which is related to a collapsing variant of FSGS. Nevertheless, other viruses are also suspected for causing a collapsing or noncollapsing variant, for example, hepatitis B virus, parvovirus B19, and Cytomegalovirus. Although the systemic infection mechanism is different for these viruses, there are similarities in the pathomechanism for the induction of FSGS. As the podocyte is the key structure in the pathogenesis of FSGS, a direct infection of these cells or immediate damage through the virus or viral components has to be considered. Although viral infections are a very rare cause for FSGS in children, the treating pediatric nephrologist has to be aware of a possible underlying infection, as this has a relevant impact on therapy and prognosis.
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