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10.1007/s00392-015-0959-4 http://scihub22266oqcxt.onion/10.1007/s00392-015-0959-4 C4882343!4882343!26802018     free     free     free Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Clin+Res+Cardiol 2016 ; 105 (ä): 544-52 Nephropedia Template TP {{tp|p=26802018|t=2016. Renal artery sympathetic denervation: observations from the UK experience |pdf=|usr=}}{{26802018}} gab.com Text Renal artery sympathetic denervation: observations from the UK experience JO: Clin Res Cardiol http://www.kidney.de/pget.php?&tw=1&pmid=26802018 #FOAvip Background: Renal denervation (RDN) may lower blood pressure (BP); however, it is unclear whether medication changes may be confounding results. Furthermore, limited data exist on pattern of ambulatory blood pressure (ABP) response?particularly in those prescribed aldosterone antagonists at the time of RDN. Methods: We examined all patients treated with RDN for treatment-resistant hypertension in 18 UK centres. Results: Results from 253 patients treated with five technologies are shown. Pre-procedural mean office BP (OBP) was 185/102 mmHg (SD 26/19; n = 253) and mean daytime ABP was 170/98 mmHg (SD 22/16; n = 186). Median number of antihypertensive drugs was 5.0: 96 % ACEi/ARB; 86 % thiazide/loop diuretic and 55 % aldosterone antagonist. OBP, available in 90 % at 11 months follow-up, was 163/93 mmHg (reduction of 22/9 mmHg). ABP, available in 70 % at 8.5 months follow-up, was 158/91 mmHg (fall of 12/7 mmHg). Mean drug changes post RDN were: 0.36 drugs added, 0.91 withdrawn. Dose changes appeared neutral. Quartile analysis by starting ABP showed mean reductions in systolic ABP after RDN of: 0.4; 6.5; 14.5 and 22.1 mmHg, respectively (p < 0.001 for trend). Use of aldosterone antagonist did not predict response (p > 0.2). Conclusion: In 253 patients treated with RDN, office BP fell by 22/9 mmHg. Ambulatory BP fell by 12/7 mmHg, though little response was seen in the lowermost quartile of starting blood pressure. Fall in BP was not explained by medication changes and aldosterone antagonist use did not affect response. Electronic supplementary material: The online version of this article (doi:10.1007/s00392-015-0959-4) contains supplementary material, which is available to authorized users. Twit Text FOAVip Renal artery sympathetic denervation: observations from the UK experience ????Clin Res Cardiol http://www.kidney.de/pget.php?&tw=1&pmid=26802018 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26802018 #FOAvip English Wikipedia <ref>{{Cite pmid|26802018}}</ref> Renal artery sympathetic denervation: observations from the UK experience #MMPMID26802018 Sharp ASP; Davies JE; Lobo MD; Bent CL; Mark PB; Burchell AE; Thackray SD; Martin U; McKane WS; Gerber RT; Wilkinson JR; Antonios TF; Doulton TW; Patterson T; Clifford PC; Lindsay A; Houston GJ; Freedman J; Das N; Belli AM; Faris M; Cleveland TJ; Nightingale AK; Hameed A; Mahadevan K; Finegold JA; Mather AN; Levy T; D?Souza R; Riley P; Moss JG; Di Mario C; Redwood SR; Baumbach A; Caulfield MJ; Dasgupta I Clin Res Cardiol 2016[]; 105 (ä): 544-52 PMID26802018show ga Background: Renal denervation (RDN) may lower blood pressure (BP); however, it is unclear whether medication changes may be confounding results. Furthermore, limited data exist on pattern of ambulatory blood pressure (ABP) response?particularly in those prescribed aldosterone antagonists at the time of RDN. Methods: We examined all patients treated with RDN for treatment-resistant hypertension in 18 UK centres. Results: Results from 253 patients treated with five technologies are shown. Pre-procedural mean office BP (OBP) was 185/102 mmHg (SD 26/19; n = 253) and mean daytime ABP was 170/98 mmHg (SD 22/16; n = 186). Median number of antihypertensive drugs was 5.0: 96 % ACEi/ARB; 86 % thiazide/loop diuretic and 55 % aldosterone antagonist. OBP, available in 90 % at 11 months follow-up, was 163/93 mmHg (reduction of 22/9 mmHg). ABP, available in 70 % at 8.5 months follow-up, was 158/91 mmHg (fall of 12/7 mmHg). Mean drug changes post RDN were: 0.36 drugs added, 0.91 withdrawn. Dose changes appeared neutral. Quartile analysis by starting ABP showed mean reductions in systolic ABP after RDN of: 0.4; 6.5; 14.5 and 22.1 mmHg, respectively (p < 0.001 for trend). Use of aldosterone antagonist did not predict response (p > 0.2). Conclusion: In 253 patients treated with RDN, office BP fell by 22/9 mmHg. Ambulatory BP fell by 12/7 mmHg, though little response was seen in the lowermost quartile of starting blood pressure. Fall in BP was not explained by medication changes and aldosterone antagonist use did not affect response. Electronic supplementary material: The online version of this article (doi:10.1007/s00392-015-0959-4) contains supplementary material, which is available to authorized users. äRenal artery sympathetic denervation: observations from the UK experie(epmc).pdf DeepDyve Pubget Overpricing