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10.2174/1570159X14666151230110058 http://scihub22266oqcxt.onion/10.2174/1570159X14666151230110058 C4876594!4876594!26714583     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Curr+Neuropharmacol 2016 ; 14 (4): 392-402 Nephropedia Template TP {{tp|p=26714583|t=2016. Molecular, Cellular and Clinical Aspects of Intracerebral Hemorrhage: Are the Enemies Within?|pdf=|usr=}}{{26714583}} gab.com Text Molecular, Cellular and Clinical Aspects of Intracerebral Hemorrhage: Are the Enemies Within JO: Curr Neuropharmacol http://www.kidney.de/pget.php?&tw=1&pmid=26714583 #FOAvip Hemorrhagic stroke is a disease with high incidence and mortality rates. In addition to the mass lesions that result from hemorrhagic stroke, substances such as the blood-derived products (BDP) (hemoglobin (Hb), heme and iron) induce a potent inflammatory response and exert direct toxic effects on neurons, astrocytes, and microglia. In the present review, we discuss the mechanisms of brain injury secondary to hemorrhagic stroke, focusing on the involvement of BDP as major players of cellular redox imbalance, inflammation, and glutamate excitotoxicity. Potential natural mechanisms of protection against free Hb and heme such as haptoglobin and hemopexin, respectively, are highlighted. We finally discuss the experimental and clinical trials targeting free iron and heme scavenging as well as inflammation, as potential new therapies to minimize the devastating effects of hemorrhagic stroke on brain structure and function. Twit Text FOAVip Molecular, Cellular and Clinical Aspects of Intracerebral Hemorrhage: Are the Enemies Within ????Curr Neuropharmacol http://www.kidney.de/pget.php?&tw=1&pmid=26714583 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26714583 #FOAvip English Wikipedia <ref>{{Cite pmid|26714583}}</ref> Molecular, Cellular and Clinical Aspects of Intracerebral Hemorrhage: Are the Enemies Within? #MMPMID26714583 Righy C; Bozza MT; Oliveira MF; Bozza FA Curr Neuropharmacol 2016[May]; 14 (4): 392-402 PMID26714583show ga Hemorrhagic stroke is a disease with high incidence and mortality rates. In addition to the mass lesions that result from hemorrhagic stroke, substances such as the blood-derived products (BDP) (hemoglobin (Hb), heme and iron) induce a potent inflammatory response and exert direct toxic effects on neurons, astrocytes, and microglia. In the present review, we discuss the mechanisms of brain injury secondary to hemorrhagic stroke, focusing on the involvement of BDP as major players of cellular redox imbalance, inflammation, and glutamate excitotoxicity. Potential natural mechanisms of protection against free Hb and heme such as haptoglobin and hemopexin, respectively, are highlighted. We finally discuss the experimental and clinical trials targeting free iron and heme scavenging as well as inflammation, as potential new therapies to minimize the devastating effects of hemorrhagic stroke on brain structure and function. äMolecular, Cellular and Clinical Aspects of Intracerebral Hemorrhage: (epmc).pdf DeepDyve Pubget Overpricing