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10.1086/686011 http://scihub22266oqcxt.onion/10.1086/686011 C4869917!4869917!27252839     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Pulm+Circ 2016 ; 6 (2): 145-54 Nephropedia Template TP {{tp|p=27252839|t=2016. Current concepts in the pathogenesis of chronic thromboembolic pulmonary hypertension |pdf=|usr=}}{{27252839}} gab.com Text Current concepts in the pathogenesis of chronic thromboembolic pulmonary hypertension JO: Pulm Circ http://www.kidney.de/pget.php?&tw=1&pmid=27252839 #FOAvip AbstractChronic thromboembolic pulmonary hypertension (CTEPH) is characterized by fibrotic obstruction of the proximal pulmonary arteries, and it is believed to result from incomplete thrombus resolution after acute pulmonary embolism. While treatment for this condition with surgery and medical therapy has improved outcomes, our understanding of the molecular mechanisms underlying CTEPH is incomplete. Numerous risk factors have been associated with the development of CTEPH, including but not limited to acquired thrombophilias and chronic inflammatory states. A minority of patients with CTEPH have an abnormal fibrin structure that may delay thrombus resolution. Recently, examination of resected scar material in patients with CTEPH has suggested that deficient angiogenesis may play a role in thrombus nonresolution, and there is increasing interest in factors that drive intravascular scar formation. An additional challenge in CTEPH research is understanding the etiology and implications of the small-vessel disease present in many patients. Future work will likely be directed at understanding the pathways important to disease pathogenesis through further examinations of resected tissue material, continued work on animal models, and genomic approaches to identify alterations in gene expression or gene variants that may distinguish CTEPH from other forms of pulmonary hypertension. Twit Text FOAVip Current concepts in the pathogenesis of chronic thromboembolic pulmonary hypertension ????Pulm Circ http://www.kidney.de/pget.php?&tw=1&pmid=27252839 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27252839 #FOAvip English Wikipedia <ref>{{Cite pmid|27252839}}</ref> Current concepts in the pathogenesis of chronic thromboembolic pulmonary hypertension #MMPMID27252839 Matthews DT; Hemnes AR Pulm Circ 2016[Jun]; 6 (2): 145-54 PMID27252839show ga AbstractChronic thromboembolic pulmonary hypertension (CTEPH) is characterized by fibrotic obstruction of the proximal pulmonary arteries, and it is believed to result from incomplete thrombus resolution after acute pulmonary embolism. While treatment for this condition with surgery and medical therapy has improved outcomes, our understanding of the molecular mechanisms underlying CTEPH is incomplete. Numerous risk factors have been associated with the development of CTEPH, including but not limited to acquired thrombophilias and chronic inflammatory states. A minority of patients with CTEPH have an abnormal fibrin structure that may delay thrombus resolution. Recently, examination of resected scar material in patients with CTEPH has suggested that deficient angiogenesis may play a role in thrombus nonresolution, and there is increasing interest in factors that drive intravascular scar formation. An additional challenge in CTEPH research is understanding the etiology and implications of the small-vessel disease present in many patients. Future work will likely be directed at understanding the pathways important to disease pathogenesis through further examinations of resected tissue material, continued work on animal models, and genomic approaches to identify alterations in gene expression or gene variants that may distinguish CTEPH from other forms of pulmonary hypertension. äCurrent concepts in the pathogenesis of chronic thromboembolic pulmona(epmc).pdf DeepDyve Pubget Overpricing